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T淋巴细胞对致病性“许可”过程的新见解。

Shedding New Light on the Process of "Licensing" for Pathogenicity by Th Lymphocytes.

作者信息

Tan Cuiyan, Wandu Wambui S, Lee R Steven, Hinshaw Samuel H, Klinman Dennis M, Wawrousek Eric, Gery Igal

机构信息

Laboratory of Immunology, National Eye Institute, National Institutes of Health, Bethesda, MD 20892.

Genetic Engineering Core, National Eye Institute, National Institutes of Health, Bethesda, MD 20892; and.

出版信息

J Immunol. 2017 Jan 15;198(2):681-690. doi: 10.4049/jimmunol.1502108. Epub 2016 Dec 16.

Abstract

Th cells sensitized against autoantigens acquire pathogenicity following two sequential events, namely activation by their target Ag and a process named "licensing." In this study, we analyzed these processes in a transgenic mouse system in which TCR-transgenic Th cells specific to hen egg lysozyme (HEL) are adoptively transferred to recipients and induce inflammation in eyes expressing HEL. Our data show that the notion that the lung is the organ where "licensing" for pathogenicity takes place is based on biased data collected with cells injected i.v., a route in which most transferred cells enter via the lung. Thus, we found that when donor cells were activated in vitro and injected intraperitoneally, or were activated in vivo, they migrated simultaneously to the lung, spleen, and other tested organs. In all, tested organs donor cells undergo "licensing" for pathogenicity, consisting of vigorous increase in number and changes in expression levels of inflammation-related genes, monitored by both flow cytometry and microarray analysis. After reaching peak numbers, around day 3, the "licensed" donor cells migrate to the circulation and initiate inflammation in the HEL-expressing recipient eyes. Importantly, the kinetics of increase in number and of changes in gene expression by the donor cells were similar in lung, spleen, and other tested organs of the recipient mice. Furthermore, the total numbers of donor cells in the spleen at their peaks were 10- to 100-fold larger in the spleen than in the lung, contradicting the notion that the lung is the organ where "licensing" takes place.

摘要

对自身抗原致敏的Th细胞在经历两个连续事件后获得致病性,即被其靶抗原激活以及一个名为“许可”的过程。在本研究中,我们在一个转基因小鼠系统中分析了这些过程,在该系统中,将对鸡卵溶菌酶(HEL)特异的TCR转基因Th细胞过继转移至受体,并在表达HEL的眼睛中诱导炎症。我们的数据表明,认为肺是发生致病性“许可”的器官这一观点是基于通过静脉注射细胞收集的有偏差的数据,在该途径中,大多数转移细胞经肺进入。因此,我们发现当供体细胞在体外被激活并腹腔注射,或在体内被激活时,它们会同时迁移至肺、脾和其他测试器官。总之,在所有测试器官中,供体细胞都会经历致病性“许可”,包括细胞数量的急剧增加以及炎症相关基因表达水平的变化,这通过流式细胞术和微阵列分析进行监测。在达到峰值数量后,大约在第3天,“获得许可”的供体细胞迁移至循环系统并在表达HEL的受体眼睛中引发炎症。重要的是,受体小鼠的肺、脾和其他测试器官中,供体细胞数量增加的动力学以及基因表达变化的动力学是相似的。此外,供体细胞在脾脏中达到峰值时的总数比在肺中多10到100倍,这与肺是发生“许可”的器官这一观点相矛盾。

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