Ezure K, Manabe M, Otake K
Department of Neurobiology, Tokyo Metropolitan Institute for Neurosciences, Japan.
Neurosci Lett. 1989 Oct 9;104(3):303-8. doi: 10.1016/0304-3940(89)90593-4.
In Nembutal-anesthetized and artificially ventilated cats, we studied the connectivity of burst inspiratory (I) neurons in the Bötzinger complex and the ventral respiratory group (VRG) with spike-triggered averaging methods. Burst I neurons exhibited tonic (I-TON) or decrementing (I-DEC) firing patterns. Spikes of I-TON neurons induced monosynaptic EPSPs in intracellularly recorded I neurons of both the VRG and the dorsal respiratory group (DRG). Spikes of I-DEC neurons induced monosynaptic inhibitory postsynaptic potentials (IPSPs) in both VRG and DRG I neurons.
在戊巴比妥麻醉并人工通气的猫身上，我们采用触发尖峰平均法研究了博茨inger复合体和腹侧呼吸组（VRG）中爆发性吸气（I）神经元的连接性。爆发性I神经元表现出紧张性（I-TON）或递减性（I-DEC）放电模式。I-TON神经元的尖峰在VRG和背侧呼吸组（DRG）的细胞内记录的I神经元中诱发单突触兴奋性突触后电位（EPSP）。I-DEC神经元的尖峰在VRG和DRG的I神经元中均诱发单突触抑制性突触后电位（IPSP）。
