Nitric oxide synthase and polycystic kidney disease.
作者信息
Wiwanitkit Viroj
机构信息
Surin Rajabhat University, Surin, Thailand.
出版信息
J Nephropharmacol. 2015 Jun 20;4(2):85-87. eCollection 2015.
Abstract
摘要
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Role of endothelial nitric oxide synthase VNTR (intron 4 a/b) polymorphism on the progression of renal disease in autosomal dominant polycystic kidney disease.内皮型一氧化氮合酶VNTR（第4内含子a/b）多态性在常染色体显性多囊肾病肾脏疾病进展中的作用
J Renal Inj Prev. 2014 Jul 1;3(3):69-73. doi: 10.12861/jrip.2014.21. eCollection 2014.
2
Histone deacetylase 1 reduces NO production in endothelial cells via lysine deacetylation of NO synthase 3.组蛋白去乙酰化酶 1 通过赖氨酸去乙酰化作用降低内皮细胞中一氧化氮合酶 3 的产生。
Am J Physiol Heart Circ Physiol. 2014 Sep 1;307(5):H803-9. doi: 10.1152/ajpheart.00243.2014. Epub 2014 Jul 11.
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NOS3 tagSNPs does not modify the chronic kidney disease progression in autosomal dominant polycystic kidney disease.一氧化氮合酶3标签单核苷酸多态性不会改变常染色体显性多囊肾病中的慢性肾病进展。
Nephrology (Carlton). 2014 Sep;19(9):537-41. doi: 10.1111/nep.12278.
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Fluid transport and cystogenesis in autosomal dominant polycystic kidney disease.常染色体显性多囊肾病中的液体转运与囊肿形成
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The cell biology of polycystic kidney disease.多囊肾病的细胞生物学。
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