更正：尽管适应性免疫增强，但树突状细胞中I型干扰素受体缺陷促进了全身性小鼠诺如病毒的持续存在。
Correction: Type I Interferon Receptor Deficiency in Dendritic Cells Facilitates Systemic Murine Norovirus Persistence Despite Enhanced Adaptive Immunity.
作者信息
Nice Timothy J, Osborne Lisa C, Tomov Vesselin T, Artis David, Wherry E John, Virgin Herbert W
出版信息
PLoS Pathog. 2017 Feb 17;13(2):e1006223. doi: 10.1371/journal.ppat.1006223. eCollection 2017 Feb.
Abstract
[This corrects the article DOI: 10.1371/journal.ppat.1005684.].
摘要
[本文更正了文章的数字对象标识符：10.1371/journal.ppat.1005684。]
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本文引用的文献
1
Type I Interferon Receptor Deficiency in Dendritic Cells Facilitates Systemic Murine Norovirus Persistence Despite Enhanced Adaptive Immunity.树突状细胞中I型干扰素受体缺陷促进系统性小鼠诺如病毒持续存在，尽管适应性免疫增强。
PLoS Pathog. 2016 Jun 21;12(6):e1005684. doi: 10.1371/journal.ppat.1005684. eCollection 2016 Jun.
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