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On the interrelationship between virus concentration and pathological changes of pancreas in mice induced by EMC-virus.关于脑心肌炎病毒诱导的小鼠胰腺病毒浓度与病理变化之间的相互关系
Nihon Ika Daigaku Zasshi. 1987 Oct;54(5):568-9. doi: 10.1272/jnms1923.54.568.
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Animal model of human disease: diabetes mellitus (juvenile- and maturity- onset types).人类疾病动物模型：糖尿病（青少年发病型和成年发病型）。
Am J Pathol. 1975 Mar;78(3):537-40.
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A genetically determined host factor controlling susceptibility to encephalomyocarditis virus-induced diabetes in mice.一种控制小鼠对脑心肌炎病毒诱导的糖尿病易感性的基因决定宿主因子。
J Gen Virol. 1993 Jun;74 ( Pt 6):1207-13. doi: 10.1099/0022-1317-74-6-1207.
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Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.Hck在小鼠脑心肌炎病毒诱导的糖尿病发病机制中的作用。
J Virol. 2001 Feb;75(4):1949-57. doi: 10.1128/JVI.75.4.1949-1957.2001.
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Characterization of insulin-dependent diabetes mellitus induced by a new variant (DK-27) of encephalomyocarditis virus in DBA/2 mice.脑心肌炎病毒新变种（DK-27）诱导的DBA/2小鼠胰岛素依赖型糖尿病的特征分析。
Exp Anim. 1995 Jul;44(3):211-8. doi: 10.1538/expanim.44.211.
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Encephalomyocarditis virus-induced diabetes mellitus in mice: model of viral pathogenesis.
Rev Infect Dis. 1987 Sep-Oct;9(5):917-24. doi: 10.1093/clinids/9.5.917.
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The significance of T cells, B cells, antibodies and macrophages against encephalomyocarditis (EMC)-D virus-induced diabetes in mice.T细胞、B细胞、抗体和巨噬细胞在小鼠抗脑心肌炎（EMC）-D病毒诱导的糖尿病中的意义。
Arch Virol. 2008;153(7):1223-31. doi: 10.1007/s00705-008-0106-x. Epub 2008 May 24.
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Depletion of Mac1-positive macrophages protects DBA/2 mice from encephalomyocarditis virus-induced myocarditis and diabetes.Mac1阳性巨噬细胞的耗竭可保护DBA/2小鼠免受脑心肌炎病毒诱导的心肌炎和糖尿病的侵害。
J Gen Virol. 1996 Apr;77 ( Pt 4):737-41. doi: 10.1099/0022-1317-77-4-737.
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In vivo administration of serum thymic factor (FTS) prevents EMC-D virus-induced diabetes and myocarditis in BALB/cAJcl mice.在BALB/cAJcl小鼠体内给予血清胸腺因子（FTS）可预防脑心肌炎病毒（EMC-D）诱导的糖尿病和心肌炎。
Arch Virol. 1996;141(1):73-83. doi: 10.1007/BF01718589.
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