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PCYT1A基因的突变是导致孤立性视网膜营养不良的原因。

Mutations in the PCYT1A gene are responsible for isolated forms of retinal dystrophy.

作者信息

Testa Francesco, Filippelli Mariaelena, Brunetti-Pierri Raffaella, Di Fruscio Giuseppina, Di Iorio Valentina, Pizzo Mariateresa, Torella Annalaura, Barillari Maria Rosaria, Nigro Vincenzo, Brunetti-Pierri Nicola, Simonelli Francesca, Banfi Sandro

机构信息

Eye Clinic, Multidisciplinary Department of Medical, Surgical and Dental Sciences, Università degli Studi della Campania 'Luigi Vanvitelli', Naples, Italy.

Medical Genetics, Department of Biochemistry, Biophysics and General Pathology, Università degli Studi della Campania 'Luigi Vanvitelli', Naples, Italy.

出版信息

Eur J Hum Genet. 2017 May;25(5):651-655. doi: 10.1038/ejhg.2017.23. Epub 2017 Mar 8.

Abstract

Mutations in the PCYT1A gene have been recently linked to two different phenotypes: one characterized by spondylometaphyseal dysplasia and cone-rod dystrophy (SMD-CRD) and the other by congenital lipodystrophy, severe fatty liver disease, and reduced HDL cholesterol without any retinal or skeletal involvement. Here, we identified, by next generation sequencing, sequence variants affecting function in the PCYT1A gene in three young patients with isolated retinal dystrophy from two different Italian families. A thorough clinical evaluation of the patients, with whole skeleton X-ray, metabolic assessment and liver ultrasound failed to reveal signs of skeletal dysplasia, metabolic and hepatic alterations. This is the first report showing that the PCYT1A gene can be responsible for isolated forms of retinal dystrophy, particularly without any skeletal involvement, thus further expanding the phenotypic spectrum induced by mutations in this gene.

摘要

PCYT1A基因的突变最近与两种不同的表型相关:一种以脊椎干骺端发育不良和圆锥-杆状营养不良(SMD-CRD)为特征,另一种以先天性脂肪营养不良、严重脂肪肝疾病和高密度脂蛋白胆固醇降低为特征,且无任何视网膜或骨骼受累。在这里,我们通过下一代测序在来自两个不同意大利家庭的三名孤立性视网膜营养不良的年轻患者中鉴定出影响PCYT1A基因功能的序列变异。对患者进行全面的临床评估,包括全骨骼X线检查、代谢评估和肝脏超声检查,均未发现骨骼发育不良、代谢和肝脏改变的迹象。这是第一份报告表明PCYT1A基因可能是孤立性视网膜营养不良的病因,尤其是在无任何骨骼受累的情况下,从而进一步扩大了该基因突变所诱导的表型谱。

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