The real culprit behind diabetic nephropathy: impaired renal autoregulation?
作者信息
Liu Ruisheng
机构信息
Department of Molecular Pharmacology & Physiology, University of South Florida College of Medicine, Tampa, Florida.
出版信息
Physiol Rep. 2017 Mar;5(5). doi: 10.14814/phy2.13138.
Abstract
摘要
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The real culprit behind diabetic nephropathy: impaired renal autoregulation?糖尿病肾病背后的真正元凶：肾自动调节功能受损？
Physiol Rep. 2017 Mar;5(5). doi: 10.14814/phy2.13138.
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本文引用的文献
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Impaired myogenic response of the afferent arteriole contributes to the increased susceptibility to renal disease in Milan normotensive rats.传入小动脉的肌源性反应受损导致米兰正常血压大鼠对肾脏疾病的易感性增加。
Physiol Rep. 2017 Feb;5(3). doi: 10.14814/phy2.13089.
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The Prevalence and Management of Diabetic Nephropathy in Asia.亚洲地区糖尿病肾病的患病率和管理。
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Macula Densa Nitric Oxide Synthase 1β Protects against Salt-Sensitive Hypertension.致密斑一氧化氮合酶1β可预防盐敏感性高血压。
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Renal autoregulation in health and disease.健康与疾病状态下的肾自动调节
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The diabetes control and complications trial/epidemiology of diabetes interventions and complications study at 30 years: overview.糖尿病控制与并发症试验/糖尿病干预与并发症研究30年回顾：概述
Diabetes Care. 2014;37(1):9-16. doi: 10.2337/dc13-2112.
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Genetic associations in diabetic nephropathy.糖尿病肾病的遗传相关性。
Clin Exp Nephrol. 2014 Apr;18(2):197-200. doi: 10.1007/s10157-013-0874-9. Epub 2013 Oct 16.
7
Molecular mechanisms of renal blood flow autoregulation.肾血流自身调节的分子机制。
Curr Vasc Pharmacol. 2014;12(6):845-58. doi: 10.2174/15701611113116660149.
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Characterization of the development of renal injury in Type-1 diabetic Dahl salt-sensitive rats.1 型糖尿病 Dahl 盐敏感大鼠肾损伤发展的特征描述。
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Genetic basis of the impaired renal myogenic response in FHH rats.家族性高血压性肥大症大鼠肾脏肌源性反应受损的遗传基础。
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Salt-sensitive splice variant of nNOS expressed in the macula densa cells.在致密斑细胞中表达的 nNOS 的盐敏感剪接变体。
Am J Physiol Renal Physiol. 2010 Jun;298(6):F1465-71. doi: 10.1152/ajprenal.00650.2009. Epub 2010 Mar 24.
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