脱髓鞘疾病的体内和体外模型，XXI：大鼠少突胶质细胞分化与JHMV复制控制之间的关系。 - Suppr | 超能文献

In vivo and in vitro models of demyelinating disease, XXI: relationship between differentiation of rat oligodendrocytes and control of JHMV replication.

作者信息

Beushausen S, Dales S

机构信息

Department of Microbiology and Immunology, University of Western Ontario, London, Canada.

出版信息

Adv Exp Med Biol. 1987;218:239-54. doi: 10.1007/978-1-4684-1280-2_29.

DOI:10.1007/978-1-4684-1280-2_29

Abstract

摘要

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引用本文的文献

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In vivo and in vitro models of demyelinating disease: efficiency of virus spread and formation of infectious centers among glial cells is genetically determined by the murine host.脱髓鞘疾病的体内和体外模型：病毒传播效率以及神经胶质细胞中感染中心的形成由小鼠宿主基因决定。

J Virol. 1988 Sep;62(9):3371-7. doi: 10.1128/JVI.62.9.3371-3377.1988.

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In vivo and in vitro models of demyelinating diseases. XXIV. The infectious process in cyclosporin A treated Wistar Lewis rats inoculated with JHM virus.脱髓鞘疾病的体内和体外模型。二十四。用JHM病毒接种环孢素A处理的Wistar Lewis大鼠中的感染过程。

Microb Pathog. 1989 Jan;6(1):7-16. doi: 10.1016/0882-4010(89)90003-x.