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1
Correction: Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.更正:抑制烟酰胺腺嘌呤二核苷酸磷酸氧化酶2(NOX2)可预防饱和脂肪引起的心肌细胞氧化应激和线粒体异常。
PLoS One. 2017 Mar 20;12(3):e0174525. doi: 10.1371/journal.pone.0174525. eCollection 2017.
2
Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.抑制烟酰胺腺嘌呤二核苷酸磷酸氧化酶2(NOX2)可预防饱和脂肪引起的心肌细胞氧化应激和线粒体异常。
PLoS One. 2016 Jan 12;11(1):e0145750. doi: 10.1371/journal.pone.0145750. eCollection 2016.
3
Nobiletin, a Polymethoxy Flavonoid, Protects Against Cardiac Hypertrophy Induced by Pressure-Overload via Inhibition of NAPDH Oxidases and Endoplasmic Reticulum Stress.川陈皮素,一种多甲氧基黄酮,通过抑制NADPH氧化酶和内质网应激来预防压力超负荷诱导的心肌肥大。
Cell Physiol Biochem. 2017;42(4):1313-1325. doi: 10.1159/000478960. Epub 2017 Jul 13.
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Correction: Inhibition of p38 MAPK Signaling Augments Skin Tumorigenesis via NOX2 Driven ROS Generation.更正:抑制p38丝裂原活化蛋白激酶信号通路通过NOX2驱动的活性氧生成增强皮肤肿瘤发生。
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Correction: Functional abnormalities in induced Pluripotent Stem Cell-derived cardiomyocytes generated from titin-mutated patients with dilated cardiomyopathy.更正:由患有扩张型心肌病的肌联蛋白突变患者诱导多能干细胞衍生的心肌细胞中的功能异常。
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Correction: AarF Domain Containing Kinase 3 (ADCK3) Mutant Cells Display Signs of Oxidative Stress, Defects in Mitochondrial Homeostasis and Lysosomal Accumulation.更正:含AarF结构域激酶3(ADCK3)突变的细胞表现出氧化应激迹象、线粒体稳态缺陷和溶酶体积累。
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PLoS One. 2016 Apr 26;11(4):e0154651. doi: 10.1371/journal.pone.0154651. eCollection 2016.
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In vitro treatment of HepG2 cells with saturated fatty acids reproduces mitochondrial dysfunction found in nonalcoholic steatohepatitis.用饱和脂肪酸对HepG2细胞进行体外处理,可重现非酒精性脂肪性肝炎中发现的线粒体功能障碍。
Dis Model Mech. 2015 Feb;8(2):183-91. doi: 10.1242/dmm.018234. Epub 2014 Dec 24.
9
Correction: The Interaction of the Atm Genotype with Inflammation and Oxidative Stress.更正:Atm基因型与炎症和氧化应激的相互作用。
PLoS One. 2015 Jun 17;10(6):e0130645. doi: 10.1371/journal.pone.0130645. eCollection 2015.
10
Correction: FoxO3a Modulates Hypoxia Stress Induced Oxidative Stress and Apoptosis in Cardiac Microvascular Endothelial Cells.更正:FoxO3a调节缺氧应激诱导的心脏微血管内皮细胞氧化应激和细胞凋亡。
PLoS One. 2013 Dec 16;8(12). doi: 10.1371/annotation/1c419628-f1b5-45de-9f8a-43f834309ebb. eCollection 2013.

引用本文的文献

1
Thyroid hormone increases fatty acid use in fetal ovine cardiac myocytes.甲状腺激素增加胎羊心肌细胞脂肪酸的利用。
Physiol Rep. 2023 Nov;11(22):e15865. doi: 10.14814/phy2.15865.

本文引用的文献

1
Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.抑制烟酰胺腺嘌呤二核苷酸磷酸氧化酶2(NOX2)可预防饱和脂肪引起的心肌细胞氧化应激和线粒体异常。
PLoS One. 2016 Jan 12;11(1):e0145750. doi: 10.1371/journal.pone.0145750. eCollection 2016.

更正:抑制烟酰胺腺嘌呤二核苷酸磷酸氧化酶2(NOX2)可预防饱和脂肪引起的心肌细胞氧化应激和线粒体异常。

Correction: Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.

作者信息

Joseph Leroy C, Barca Emanuele, Subramanyam Prakash, Komrowski Michael, Pajvani Utpal, Colecraft Henry M, Hirano Michio, Morrow John P

出版信息

PLoS One. 2017 Mar 20;12(3):e0174525. doi: 10.1371/journal.pone.0174525. eCollection 2017.

DOI:10.1371/journal.pone.0174525
PMID:28319186
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5358878/
Abstract

[This corrects the article DOI: 10.1371/journal.pone.0145750.].

摘要

[本文更正了文章的数字对象标识符:10.1371/journal.pone.0145750。]