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Inflammation Effects on Stent Restenosis.

作者信息

Kucukseymen Selcuk

机构信息

1 Department of Cardiology, University of Health Sciences, Antalya Education and Research Hospital, Antalya, Turkey.

出版信息

Angiology. 2017 Sep;68(8):741. doi: 10.1177/0003319717701659. Epub 2017 Mar 30.

DOI:10.1177/0003319717701659
PMID:28359157
Abstract
摘要

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引用本文的文献

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Multilevel Assessment of Stent-Induced Inflammation in the Adjacent Vascular Tissue.血管组织支架置入后炎症的多层次评估。
ACS Biomater Sci Eng. 2023 Aug 14;9(8):4747-4760. doi: 10.1021/acsbiomaterials.3c00540. Epub 2023 Jul 21.
2
Silencing of FOS-like antigen 1 represses restenosis the ERK/AP-1 pathway in type 2 diabetic mice.沉默 FOS 样抗原 1 抑制 2 型糖尿病小鼠的再狭窄——ERK/AP-1 通路。
Diab Vasc Dis Res. 2021 Nov-Dec;18(6):14791641211058855. doi: 10.1177/14791641211058855.
3
The number of stents was an independent risk of stent restenosis in patients undergoing percutaneous coronary intervention.
在接受经皮冠状动脉介入治疗的患者中，支架数量是支架再狭窄的独立危险因素。
Medicine (Baltimore). 2019 Dec;98(50):e18312. doi: 10.1097/MD.0000000000018312.
4
Tumor necrosis factor alpha-stimulated gene-6 (TSG-6) inhibits the inflammatory response by inhibiting the activation of P38 and JNK signaling pathway and decreases the restenosis of vein grafts in rats.肿瘤坏死因子α刺激基因-6（TSG-6）通过抑制P38和JNK信号通路的激活来抑制炎症反应，并降低大鼠静脉移植物的再狭窄。
Heart Vessels. 2017 Dec;32(12):1536-1545. doi: 10.1007/s00380-017-1059-3. Epub 2017 Oct 3.