Laurberg S, Swash M, Snooks S J, Henry M M
Sir Alan Parks Physiology Unit, St Mark's Hospital, London, England.
Arch Neurol. 1988 Nov;45(11):1250-3. doi: 10.1001/archneur.1988.00520350088021.
The relationship between the pudendal and perineal nerve terminal motor latencies and descent (weakness) of the perineum on straining was investigated in 31 patients with idiopathic fecal incontinence, and in 30 patients with double incontinence. Pelvic floor descent was correlated with increased pudendal nerve terminal motor latency in both groups of patients. In the patients with double incontinence, there was a less significant correlation between perineal descent and increased perineal nerve terminal motor latency. In the patients with fecal incontinence, but without urinary incontinence, there was no correlation between perineal descent and perineal nerve terminal motor latency. These data support the concept that pelvic floor weakness can result in damage to the pudendal and perineal nerves, leading to fecal and urinary incontinence. In patients with isolated fecal incontinence the perineal nerves are relatively spared. Thus these common types of incontinence probably have a neurologic cause, and neurophysiologic methods can be used in their assessment.
在31例特发性大便失禁患者和30例大小便失禁患者中，研究了阴部神经和会阴神经终末运动潜伏期与用力时会阴下降（松弛）之间的关系。两组患者的盆底下降均与阴部神经终末运动潜伏期延长相关。在大小便失禁患者中，会阴下降与会阴神经终末运动潜伏期延长之间的相关性较弱。在仅有大便失禁而无尿失禁的患者中，会阴下降与会阴神经终末运动潜伏期之间无相关性。这些数据支持这样一种观点，即盆底功能减弱可导致阴部神经和会阴神经损伤，进而引起大便失禁和尿失禁。在仅有大便失禁的患者中，会阴神经相对未受影响。因此，这些常见类型的失禁可能有神经学原因，神经生理学方法可用于其评估。
