Retraction Note to: Dose-dependent changes in neuroinflammatory and arachidonic acid cascade markers with synaptic marker loss in rat lipopolysaccharide infusion model of neuroinflammation.
作者信息
Kellom Matthew, Basselin Mireille, Keleshian Vasken L, Chen Mei, Rapoport Stanley I, Rao Jagadeesh S
机构信息
Brain Physiology and Metabolism Section, National Institute on Aging, National Institutes of Health, 9000 Rockville Pike, Bldg. 9, 1S-126, Bethesda, MD, USA.
出版信息
BMC Neurosci. 2017 May 9;18(1):40. doi: 10.1186/s12868-017-0360-5.
Abstract
摘要
相似文献
1
Retraction Note to: Dose-dependent changes in neuroinflammatory and arachidonic acid cascade markers with synaptic marker loss in rat lipopolysaccharide infusion model of neuroinflammation.撤稿说明：大鼠神经炎症脂多糖输注模型中神经炎症和花生四烯酸级联标志物的剂量依赖性变化与突触标志物丢失。
BMC Neurosci. 2017 May 9;18(1):40. doi: 10.1186/s12868-017-0360-5.
2
Retraction Note to: Increased neuroinflammatory and arachidonic acid cascade markers, and reduced synaptic proteins, in brain of HIV-1 transgenic rats.撤回说明：HIV-1转基因大鼠大脑中神经炎症和花生四烯酸级联反应标志物增加，突触蛋白减少。
J Neuroinflammation. 2017 May 9;14(1):95. doi: 10.1186/s12974-017-0872-z.
3
Chronic lithium administration attenuates up-regulated brain arachidonic acid metabolism in a rat model of neuroinflammation.在神经炎症大鼠模型中，长期给予锂可减弱大脑中上调的花生四烯酸代谢。
J Neurochem. 2007 Aug;102(3):761-72. doi: 10.1111/j.1471-4159.2007.04593.x. Epub 2007 May 4.
4
Altered neuroinflammatory, arachidonic acid cascade and synaptic markers in postmortem Alzheimer's disease brain.阿尔茨海默病患者死后大脑中神经炎症、花生四烯酸级联反应及突触标志物的改变。
Transl Psychiatry. 2017 May 9;7(5):e1127. doi: 10.1038/tp.2017.97.
5
Enhanced susceptibility of S-100B transgenic mice to neuroinflammation and neuronal dysfunction induced by intracerebroventricular infusion of human beta-amyloid.S-100B转基因小鼠对脑室内注射人β-淀粉样蛋白诱导的神经炎症和神经元功能障碍的易感性增强。
Glia. 2005 Aug 15;51(3):209-16. doi: 10.1002/glia.20194.
6
Early molecular and behavioral response to lipopolysaccharide in the WAG/Rij rat model of absence epilepsy and depressive-like behavior, involves interplay between AMPK, AKT/mTOR pathways and neuroinflammatory cytokine release.早期分子和行为对 WAG/Rij 大鼠癫痫发作和抑郁样行为模型中脂多糖的反应，涉及 AMPK、AKT/mTOR 途径和神经炎性细胞因子释放之间的相互作用。
Brain Behav Immun. 2014 Nov;42:157-68. doi: 10.1016/j.bbi.2014.06.016. Epub 2014 Jul 3.
7
In vivo expression of cyclooxygenase-1 in activated microglia and macrophages during neuroinflammation visualized by PET with 11C-ketoprofen methyl ester.在神经炎症期间，通过 PET 用 11C-酮洛芬甲酯可视化观察到激活的小胶质细胞和巨噬细胞中环氧化酶-1 的体内表达。
J Nucl Med. 2011 Jul;52(7):1094-101. doi: 10.2967/jnumed.110.084046. Epub 2011 Jun 16.
8
Interleukin 1 receptor antagonist knockout mice show enhanced microglial activation and neuronal damage induced by intracerebroventricular infusion of human beta-amyloid.白细胞介素1受体拮抗剂基因敲除小鼠在脑室内注入人β-淀粉样蛋白后，显示出小胶质细胞激活增强和神经元损伤加剧。
J Neuroinflammation. 2005 Jun 20;2:15. doi: 10.1186/1742-2094-2-15.
9
In vivo imaging detects a transient increase in brain arachidonic acid metabolism: a potential marker of neuroinflammation.体内成像检测到脑花生四烯酸代谢的短暂增加：一种神经炎症的潜在标志物。
J Neurochem. 2004 Nov;91(4):936-45. doi: 10.1111/j.1471-4159.2004.02786.x.
10
Progressive dopamine neuron loss following supra-nigral lipopolysaccharide (LPS) infusion into rats exposed to LPS prenatally.向产前暴露于脂多糖（LPS）的大鼠脑黑质以上区域注入LPS后，多巴胺神经元进行性丧失。
Exp Neurol. 2006 Jun;199(2):499-512. doi: 10.1016/j.expneurol.2006.01.010. Epub 2006 Feb 28.
本文引用的文献
1
Dose-dependent changes in neuroinflammatory and arachidonic acid cascade markers with synaptic marker loss in rat lipopolysaccharide infusion model of neuroinflammation.脂多糖诱导的神经炎症大鼠模型中突触相关标志物丢失伴随神经炎症和花生四烯酸级联标志物的剂量依赖性变化。
BMC Neurosci. 2012 May 23;13:50. doi: 10.1186/1471-2202-13-50.
Zotero 插件