PRKAG2心肌病诱导多能干细胞和微组织模型的综合分析确定AMPK为代谢、存活和纤维化的调节因子。 - Suppr | 超能文献

Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and Fibrosis.

作者信息

Hinson J Travis, Chopra Anant, Lowe Andre, Sheng Calvin C, Gupta Rajat M, Kuppusamy Rajarajan, O'Sullivan John, Rowe Glenn, Wakimoto Hiroko, Gorham Joshua, Burke Michael A, Zhang Kehan, Musunuru Kiran, Gerszten Robert E, Wu Sean M, Chen Christopher S, Seidman Jonathan G, Seidman Christine E

出版信息

Cell Rep. 2017 Jun 13;19(11):2410. doi: 10.1016/j.celrep.2017.05.038.

DOI:10.1016/j.celrep.2017.05.038

Abstract

摘要

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