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Deformability of transfused red blood cells is a potent effector of transfusion-induced hemoglobin increment: A study with β-thalassemia major patients.

作者信息

Barshtein Gregory, Goldschmidt Neta, Pries Axel R, Zelig Orly, Arbell Dan, Yedgar Saul

机构信息

Department of Biochemistry, Hebrew University Faculty of Medicine, Jerusalem, Israel.

Department of Hematology, Hadassah University Hospital, Jerusalem, Israel.

出版信息

Am J Hematol. 2017 Sep;92(9):E559-E560. doi: 10.1002/ajh.24821. Epub 2017 Jul 19.

DOI:10.1002/ajh.24821
PMID:28614898
Abstract
摘要

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1
Deformability of transfused red blood cells is a potent effector of transfusion-induced hemoglobin increment: A study with β-thalassemia major patients.输注红细胞的可变形性是输血诱导血红蛋白增加的有力影响因素：一项针对重型β地中海贫血患者的研究。
Am J Hematol. 2017 Sep;92(9):E559-E560. doi: 10.1002/ajh.24821. Epub 2017 Jul 19.
2
Deformability of transfused red blood cells is a potent determinant of transfusion-induced change in recipient's blood flow.输注红细胞的可变形性是输血引起受血者血流变化的一个重要决定因素。
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RELATIONSHIP OF ERYTHROCYTE SURVIVAL AND RETICULOCYTE LEVELS TO SPLENECTOMY AND NEED FOR TRANSFUSION IN THALASSEMIA MAJOR.重型地中海贫血中红细胞存活及网织红细胞水平与脾切除术和输血需求的关系
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Red cell alloimmunisation in regularly transfused beta thalassemia patients in Pakistan.巴基斯坦定期输血的β地中海贫血患者中的红细胞同种免疫
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ERYTHROCYTE LIFE-SPAN AND SITES OF DESTRUCTION IN THALASSEMIA MAJOR. RELATION TO CLINICAL AND LABORATORY FINDINGS.重型地中海贫血中红细胞的寿命及破坏部位。与临床和实验室检查结果的关系。
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Comparison of a transfusion preparation of newly formed red cells and standard washed red cell transfusions in patients with homozygous beta-thalassemia.纯合子β地中海贫血患者中新型红细胞输血制剂与标准洗涤红细胞输血的比较。
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[Partial splenectomy in thalassemia major. Apropos of 19 cases].[重型地中海贫血的部分脾切除术。附19例报告]
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Rheological behaviour of red blood cells in beta and deltabeta thalassemia trait.β和δβ地中海贫血特征中红细胞的流变学行为。
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Platelet and not erythrocyte microparticles are procoagulant in transfused thalassaemia major patients.在重度输血性地中海贫血患者中，具有促凝血作用的是血小板微粒而非红细胞微粒。
Br J Haematol. 2015 Nov;171(4):615-24. doi: 10.1111/bjh.13609. Epub 2015 Jul 24.
10
[Hemorheologic characteristics in beta-thalassemic homozygotes before and after splenectomy].
Pediatr Med Chir. 1985 Jan-Feb;7(1):73-6.

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