• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Correction: BteA Secreted from the Bordetella bronchiseptica Type III Secetion System Induces Necrosis through an Actin Cytoskeleton Signaling Pathway and Inhibits Phagocytosis by Macrophages.更正：支气管败血波氏杆菌III型分泌系统分泌的BteA通过肌动蛋白细胞骨架信号通路诱导坏死并抑制巨噬细胞的吞噬作用。
PLoS One. 2017 Jun 22;12(6):e0180287. doi: 10.1371/journal.pone.0180287. eCollection 2017.
2
BteA Secreted from the Bordetella bronchiseptica Type III Secetion System Induces Necrosis through an Actin Cytoskeleton Signaling Pathway and Inhibits Phagocytosis by Macrophages.支气管败血波氏杆菌Ⅲ型分泌系统分泌的BteA通过肌动蛋白细胞骨架信号通路诱导坏死并抑制巨噬细胞的吞噬作用。
PLoS One. 2016 Feb 1;11(2):e0148387. doi: 10.1371/journal.pone.0148387. eCollection 2016.
3
In-vitro and in-vivo analysis of the production of the Bordetella type three secretion system effector A in Bordetella pertussis, Bordetella parapertussis and Bordetella bronchiseptica.在体外和体内分析百日咳博德特氏菌、副百日咳博德特氏菌和支气管败血博德特氏菌中产生的博德特氏菌 III 型分泌系统效应物 A。
Microbes Infect. 2013 May;15(5):399-408. doi: 10.1016/j.micinf.2013.02.006. Epub 2013 Mar 5.
4
A genome-wide screen identifies a Bordetella type III secretion effector and candidate effectors in other species.全基因组筛选鉴定出一种博德特氏菌III型分泌效应蛋白及其他物种中的候选效应蛋白。
Mol Microbiol. 2005 Oct;58(1):267-79. doi: 10.1111/j.1365-2958.2005.04823.x.
5
Differential regulation of type III secretion and virulence genes in Bordetella pertussis and Bordetella bronchiseptica by a secreted anti-σ factor.分泌型抗σ因子对百日咳博德特氏菌和支气管败血博德特氏菌III型分泌及毒力基因的差异调控
Proc Natl Acad Sci U S A. 2016 Mar 1;113(9):2341-8. doi: 10.1073/pnas.1600320113. Epub 2016 Feb 16.
6
Bordetella effector BopN is translocated into host cells via its N-terminal residues.博德特氏菌效应蛋白BopN通过其N端残基转运到宿主细胞中。
Microbiol Immunol. 2017 Jun;61(6):206-214. doi: 10.1111/1348-0421.12489.
7
Interactions of pulmonary collectins with Bordetella bronchiseptica and Bordetella pertussis lipopolysaccharide elucidate the structural basis of their antimicrobial activities.肺部凝集素与支气管败血博德特氏菌和百日咳博德特氏菌脂多糖的相互作用阐明了它们抗菌活性的结构基础。
Infect Immun. 2004 Dec;72(12):7124-30. doi: 10.1128/IAI.72.12.7124-7130.2004.
8
Correction: Actin Cytoskeleton Regulation of Epithelial Mesenchymal Transition in Metastatic Cancer Cells.更正：转移性癌细胞中上皮-间质转化的肌动蛋白细胞骨架调控
PLoS One. 2015 Jul 13;10(7):e0132759. doi: 10.1371/journal.pone.0132759. eCollection 2015.
9
Mac-1-mediated uptake and killing of Bordetella bronchiseptica by porcine alveolar macrophages.
J Vet Sci. 2003 Apr;4(1):41-9.
10
Bordetella bronchiseptica Bcr4 antagonizes the negative regulatory function of BspR via its role in type III secretion.支气管败血波氏杆菌Bcr4通过其在III型分泌中的作用拮抗BspR的负调控功能。
Microbiol Immunol. 2018 Dec;62(12):743-754. doi: 10.1111/1348-0421.12659. Epub 2018 Dec 14.

本文引用的文献

1
BteA Secreted from the Bordetella bronchiseptica Type III Secetion System Induces Necrosis through an Actin Cytoskeleton Signaling Pathway and Inhibits Phagocytosis by Macrophages.支气管败血波氏杆菌Ⅲ型分泌系统分泌的BteA通过肌动蛋白细胞骨架信号通路诱导坏死并抑制巨噬细胞的吞噬作用。
PLoS One. 2016 Feb 1;11(2):e0148387. doi: 10.1371/journal.pone.0148387. eCollection 2016.

更正：支气管败血波氏杆菌III型分泌系统分泌的BteA通过肌动蛋白细胞骨架信号通路诱导坏死并抑制巨噬细胞的吞噬作用。

Correction: BteA Secreted from the Bordetella bronchiseptica Type III Secetion System Induces Necrosis through an Actin Cytoskeleton Signaling Pathway and Inhibits Phagocytosis by Macrophages.

作者信息

Kuwae Asaomi, Momose Fumitaka, Nagamatsu Kanna, Suyama Yasuharu, Abe Akio

出版信息

PLoS One. 2017 Jun 22;12(6):e0180287. doi: 10.1371/journal.pone.0180287. eCollection 2017.

DOI:10.1371/journal.pone.0180287
PMID:28640910
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5481007/
Abstract

[This corrects the article DOI: 10.1371/journal.pone.0148387.].

摘要

[本文更正了文章的数字对象标识符：10.1371/journal.pone.0148387。]