Ali Aamna M, Ramos Christopher R, Glebova Natalia O
Arrowhead Regional Medical Center/Kaiser Fontana Department of General Surgery, Colton, CA.
Division of Vascular Surgery, Department of Surgery, Emory University School of Medicine, Atlanta, GA.
Ann Vasc Surg. 2018 Jan;46:206.e5-206.e10. doi: 10.1016/j.avsg.2017.07.023. Epub 2017 Jul 21.
Popliteal artery entrapment syndrome is an uncommon condition in which anatomic or functional popliteal artery compression causes arterial insufficiency. We present a case of popliteal entrapment with runoff thrombosis treated with suprageniculate release of entrapment without distal bypass.
A 15-year old boy with Klinefelter syndrome presented with right leg claudication severely limiting his activity. He had a palpable femoral pulse, but no palpable popliteal or foot pulses on the right. Noninvasive testing showed a partially thrombosed popliteal artery with an ankle-brachial index (ABI) of 0.69. Computed tomography scan revealed type III popliteal entrapment with distal thromboses and abnormal insertion of gastrocnemius muscle. Popliteal entrapment release was performed via a medial suprageniculate approach in consideration for distal bypass. The soleus was released first; intraoperative angiography showed continued popliteal compression with forced dorsiflexion. This was followed by release of the gastrocnemius and found caudal and medial to the soleus as a tight band. Repeat angiography showed cessation of popliteal artery compression with dorsiflexion. Bypass was not performed due to improvement of distal flow seen on angiography. Postoperative recovery was unremarkable. On 1-month and 9-month follow-up, he had a normal ABI and arterial duplex, was asymptomatic, and had returned to normal activities.
We describe suprageniculate approach to popliteal release that may be useful if a distal bypass is planned. In this case, bypass was unnecessary despite the abnormal appearance of distal runoff on preoperative imaging, as the child's perfusion improved with entrapment release alone, and arterial remodeling over time resulted in normal perfusion and arterial appearance on duplex imaging.
腘动脉受压综合征是一种罕见疾病,解剖学或功能性腘动脉受压导致动脉供血不足。我们报告一例伴有血流缓慢血栓形成的腘动脉受压病例,采用腘动脉上松解术治疗,未进行远端旁路移植术。
一名患有克兰费尔特综合征的15岁男孩出现右腿间歇性跛行,严重限制了其活动。他的股动脉搏动可触及,但右侧腘动脉和足部搏动未触及。无创检查显示腘动脉部分血栓形成,踝肱指数(ABI)为0.69。计算机断层扫描显示III型腘动脉受压伴远端血栓形成以及腓肠肌异常附着。考虑到远端旁路移植术,通过腘动脉上内侧入路进行腘动脉受压松解术。首先松解比目鱼肌;术中血管造影显示在强力背屈时腘动脉仍持续受压。随后松解腓肠肌,发现其在比目鱼肌的尾侧和内侧形成一条紧绷带。重复血管造影显示背屈时腘动脉压迫解除。由于血管造影显示远端血流改善,未进行旁路移植术。术后恢复情况良好。在1个月和9个月的随访中,他的ABI和动脉双功超声检查结果正常,无症状,已恢复正常活动。
我们描述了一种腘动脉松解的腘动脉上入路,如果计划进行远端旁路移植术,该方法可能有用。在本病例中,尽管术前影像学检查显示远端血流异常,但由于仅通过松解受压部位患儿的灌注即得到改善,且随着时间推移动脉重塑导致双功超声检查显示灌注和动脉形态正常,因此无需进行旁路移植术。