交感神经激活会减少猫肠道中血管活性肠肽的释放。
Sympathetic nerve activation decreases the release of vasoactive intestinal polypeptide from the feline intestine.
作者信息
Sjöqvist A, Fahrenkrug J
出版信息
Acta Physiol Scand. 1986 Aug;127(4):419-23. doi: 10.1111/j.1748-1716.1986.tb07922.x.
The splanchnic nerves to the small intestine were stimulated in anaesthetized cats. Activation of the sympathetic nerves caused vasoconstriction, increased net fluid absorption and decreased release of vasoactive intestinal polypeptide (VIP) in the small intestine. In the colon, parasympathetic nerve stimulation elicited vasodilatation and increased release of VIP. Additional stimulation of the sympathetic lumbar colonic nerves decreased the colonic blood flow and inhibited the release of VIP. These effects of the stimulation of the lumbar colonic nerves were blocked by phentolamine. It is concluded that, in the feline intestine, sympathetic nerve stimulation presynaptically decreased the release of VIP via an alpha-adrenergic mechanism.
在麻醉的猫身上刺激通向小肠的内脏神经。交感神经的激活导致血管收缩、肠内净液体吸收增加以及血管活性肠肽（VIP）释放减少。在结肠，副交感神经刺激引起血管舒张并增加VIP释放。额外刺激交感腰结肠神经会减少结肠血流并抑制VIP释放。酚妥拉明可阻断腰结肠神经刺激的这些效应。得出的结论是，在猫的肠道中，交感神经刺激通过α-肾上腺素能机制突触前减少VIP的释放。
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