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《关于“爱泼斯坦-巴尔病毒诱导基因3(EBI3)阻断通过双向相互调节信号转导和转录激活因子3(STAT3)信号通路诱导抗肿瘤细胞毒性T淋巴细胞反应并抑制结直肠癌肿瘤生长”的勘误》

Corrigendum to "Epstein-Barr Virus-Induced Gene 3 (EBI3) Blocking Leads to Induce Antitumor Cytotoxic T Lymphocyte Response and Suppress Tumor Growth in Colorectal Cancer by Bidirectional Reciprocal-Regulation STAT3 Signaling Pathway".

作者信息

Liang Yanfang, Chen Qianqian, Du Wenjing, Chen Can, Li Feifei, Yang Jingying, Peng Jianyu, Kang Dongping, Lin Bihua, Chai Xingxing, Zhou Keyuan, Zeng Jincheng

机构信息

Department of Pathology, Dongguan Hospital, Medical College of Jinan University, The Fifth People's Hospital of Dongguan, Dongguan 523905, China.

Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan 523808, China.

出版信息

Mediators Inflamm. 2017;2017:2917134. doi: 10.1155/2017/2917134. Epub 2017 Jul 27.

DOI:10.1155/2017/2917134
PMID:28819340
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5551508/
Abstract

[This corrects the article DOI: 10.1155/2016/3214105.].

摘要

[本文更正了文章的数字对象标识符:10.1155/2016/3214105。]

相似文献

1
Corrigendum to "Epstein-Barr Virus-Induced Gene 3 (EBI3) Blocking Leads to Induce Antitumor Cytotoxic T Lymphocyte Response and Suppress Tumor Growth in Colorectal Cancer by Bidirectional Reciprocal-Regulation STAT3 Signaling Pathway".《关于“爱泼斯坦-巴尔病毒诱导基因3(EBI3)阻断通过双向相互调节信号转导和转录激活因子3(STAT3)信号通路诱导抗肿瘤细胞毒性T淋巴细胞反应并抑制结直肠癌肿瘤生长”的勘误》
Mediators Inflamm. 2017;2017:2917134. doi: 10.1155/2017/2917134. Epub 2017 Jul 27.
2
Epstein-Barr Virus-Induced Gene 3 (EBI3) Blocking Leads to Induce Antitumor Cytotoxic T Lymphocyte Response and Suppress Tumor Growth in Colorectal Cancer by Bidirectional Reciprocal-Regulation STAT3 Signaling Pathway.爱泼斯坦-巴尔病毒诱导基因3(EBI3)阻断通过双向相互调节信号转导和转录激活因子3(STAT3)信号通路诱导抗肿瘤细胞毒性T淋巴细胞反应并抑制结直肠癌肿瘤生长。
Mediators Inflamm. 2016;2016:3214105. doi: 10.1155/2016/3214105. Epub 2016 May 10.
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First characterization of a teleost Epstein-Barr virus-induced gene 3 (EBI3) reveals a regulatory effect of EBI3 on the innate immune response of peripheral blood leukocytes.首次对一种鱼类 Epstein-Barr 病毒诱导基因 3(EBI3)进行了特征描述,揭示了 EBI3 对外周血白细胞固有免疫反应的调节作用。
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Frequent expression of the Epstein-Barr virus (EBV)-induced gene, EBI3, an IL-12 p40-related cytokine, in Hodgkin and Reed-Sternberg cells.爱泼斯坦-巴尔病毒(EBV)诱导基因EBI3(一种与IL-12 p40相关的细胞因子)在霍奇金和里德-斯腾伯格细胞中频繁表达。
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本文引用的文献

1
Epstein-Barr Virus-Induced Gene 3 (EBI3) Blocking Leads to Induce Antitumor Cytotoxic T Lymphocyte Response and Suppress Tumor Growth in Colorectal Cancer by Bidirectional Reciprocal-Regulation STAT3 Signaling Pathway.爱泼斯坦-巴尔病毒诱导基因3(EBI3)阻断通过双向相互调节信号转导和转录激活因子3(STAT3)信号通路诱导抗肿瘤细胞毒性T淋巴细胞反应并抑制结直肠癌肿瘤生长。
Mediators Inflamm. 2016;2016:3214105. doi: 10.1155/2016/3214105. Epub 2016 May 10.