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Commentary: Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?述评：蛰伏状态：3-单碘甲腺原氨酸在脑与肠之间的兴衰——从肠到脑？
Front Endocrinol (Lausanne). 2017 Aug 22;8:206. doi: 10.3389/fendo.2017.00206. eCollection 2017.
2
Erratum: Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?勘误：蛰伏：3-单碘甲腺原氨酸在脑与肠之间的兴衰——从肠到脑？
Front Endocrinol (Lausanne). 2017 Jul 10;8:158. doi: 10.3389/fendo.2017.00158. eCollection 2017.
3
Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?蛰伏：3-单碘甲腺原氨酸从脑到肠——从肠到脑的兴衰？
Front Endocrinol (Lausanne). 2017 May 31;8:118. doi: 10.3389/fendo.2017.00118. eCollection 2017.
4
In the brain of mice, 3-iodothyronamine (T1AM) is converted into 3-iodothyroacetic acid (TA1) and it is included within the signaling network connecting thyroid hormone metabolites with histamine.在小鼠大脑中，3-碘甲状腺原氨酸（T1AM）会转化为3-碘甲状腺乙酸（TA1），并且它包含在将甲状腺激素代谢物与组胺连接起来的信号网络中。
Eur J Pharmacol. 2015 Aug 15;761:130-4. doi: 10.1016/j.ejphar.2015.04.038. Epub 2015 May 1.
5
Potential role of the gut microbiota in synthetic torpor and therapeutic hypothermia.肠道微生物群在人工诱导冬眠和治疗性低温中的潜在作用。
World J Gastroenterol. 2017 Jan 21;23(3):406-413. doi: 10.3748/wjg.v23.i3.406.
6
Central Effects of 3-Iodothyronamine Reveal a Novel Role for Mitochondrial Monoamine Oxidases.3-碘甲状腺原氨酸的中枢效应揭示了线粒体单胺氧化酶的新作用。
Front Endocrinol (Lausanne). 2018 Jun 6;9:290. doi: 10.3389/fendo.2018.00290. eCollection 2018.
7
Commentary: 3-Iodothyronamine Reduces Insulin Secretion In Vitro via a Mitochondrial Mechanism.评论：3-碘甲腺原氨酸通过线粒体机制在体外减少胰岛素分泌。
Front Endocrinol (Lausanne). 2018 Feb 28;9:57. doi: 10.3389/fendo.2018.00057. eCollection 2018.
8
Alternate pathways of thyroid hormone metabolism.甲状腺激素代谢的替代途径。
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Fos-like immunoreactivity in Siberian hamster brain during initiation of torpor-like hypothermia induced by 2DG.2-脱氧葡萄糖诱导的类似蛰伏低温启动过程中叙利亚仓鼠大脑中的Fos样免疫反应性
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Tissue distribution and cardiac metabolism of 3-iodothyronamine.3-碘甲状腺原氨酸的组织分布和心脏代谢。
Endocrinology. 2010 Oct;151(10):5063-73. doi: 10.1210/en.2010-0491. Epub 2010 Aug 25.

引用本文的文献

1
The Colorful Diversity of Thyroid Hormone Metabolites.甲状腺激素代谢物的丰富多样性
Eur Thyroid J. 2019 Jun;8(3):115-129. doi: 10.1159/000497141. Epub 2019 May 21.
2
Editorial: "Homeostasis and Allostasis of Thyroid Function".社论：“甲状腺功能的稳态与应变稳态”
Front Endocrinol (Lausanne). 2018 Jun 5;9:287. doi: 10.3389/fendo.2018.00287. eCollection 2018.
3
Commentary: 3-Iodothyronamine Reduces Insulin Secretion In Vitro via a Mitochondrial Mechanism.评论：3-碘甲腺原氨酸通过线粒体机制在体外减少胰岛素分泌。
Front Endocrinol (Lausanne). 2018 Feb 28;9:57. doi: 10.3389/fendo.2018.00057. eCollection 2018.

本文引用的文献

1
In the brain of mice, 3-iodothyronamine (T1AM) is converted into 3-iodothyroacetic acid (TA1) and it is included within the signaling network connecting thyroid hormone metabolites with histamine.在小鼠大脑中，3-碘甲状腺原氨酸（T1AM）会转化为3-碘甲状腺乙酸（TA1），并且它包含在将甲状腺激素代谢物与组胺连接起来的信号网络中。
Eur J Pharmacol. 2015 Aug 15;761:130-4. doi: 10.1016/j.ejphar.2015.04.038. Epub 2015 May 1.
2
3-iodothyroacetic acid, a metabolite of thyroid hormone, induces itch and reduces threshold to noxious and to painful heat stimuli in mice.3-碘甲状腺乙酸是甲状腺激素的一种代谢产物，可诱发小鼠瘙痒并降低其对有害热刺激和疼痛热刺激的阈值。
Br J Pharmacol. 2015 Apr;172(7):1859-68. doi: 10.1111/bph.13032. Epub 2015 Jan 23.
3
Histamine mediates behavioural and metabolic effects of 3-iodothyroacetic acid, an endogenous end product of thyroid hormone metabolism.组胺介导甲状腺激素代谢的内源性终产物3-碘甲状腺乙酸的行为和代谢效应。
Br J Pharmacol. 2014 Jul;171(14):3476-84. doi: 10.1111/bph.12697.
4
Pharmacological effects of 3-iodothyronamine (T1AM) in mice include facilitation of memory acquisition and retention and reduction of pain threshold.3-碘代甲状腺素胺（T1AM）在小鼠中的药理作用包括促进记忆的获得和保持，以及降低痛阈。
Br J Pharmacol. 2013 Jan;168(2):354-62. doi: 10.1111/j.1476-5381.2012.02137.x.
5
3-Iodothyronamine: a modulator of the hypothalamus-pancreas-thyroid axes in mice.3-碘甲状腺原氨酸：调节小鼠下丘脑-胰腺-甲状腺轴的调节剂。
Br J Pharmacol. 2012 May;166(2):650-8. doi: 10.1111/j.1476-5381.2011.01823.x.

Commentary: Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?

作者信息

Laurino Annunziatina, Raimondi Laura

机构信息

Department of Psychology, Neurology, Drug Sciences, Health of the Child, Pharmacology, Section of Pharmacology and Toxicology, University of Florence, Florence, Italy.

出版信息

Front Endocrinol (Lausanne). 2017 Aug 22;8:206. doi: 10.3389/fendo.2017.00206. eCollection 2017.

DOI:10.3389/fendo.2017.00206
PMID:28878736
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5572113/
Abstract
摘要