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Marfan on the Move.

作者信息

de Waard Vivian

机构信息

Academic Medical Center, Amsterdam, The Netherlands

出版信息

J Am Heart Assoc. 2017 Sep 25;6(9):e007465. doi: 10.1161/JAHA.117.007465.

DOI:10.1161/JAHA.117.007465
PMID:28947564
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5634320/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b0f/5634320/27599ce2156a/JAH3-6-e007465-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b0f/5634320/27599ce2156a/JAH3-6-e007465-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b0f/5634320/27599ce2156a/JAH3-6-e007465-g001.jpg

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Marfan on the Move.行动中的马凡氏综合征。
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Mild aerobic exercise blocks elastin fiber fragmentation and aortic dilatation in a mouse model of Marfan syndrome associated aortic aneurysm.轻度有氧运动可阻止马凡氏综合征相关主动脉瘤小鼠模型中的弹性纤维断裂和主动脉扩张。
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Loss of elastic fiber integrity and reduction of vascular smooth muscle contraction resulting from the upregulated activities of matrix metalloproteinase-2 and -9 in the thoracic aortic aneurysm in Marfan syndrome.马凡综合征胸主动脉瘤中基质金属蛋白酶-2和-9活性上调导致弹性纤维完整性丧失和血管平滑肌收缩减弱。
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Characterization of the inflammatory cells in ascending thoracic aortic aneurysms in patients with Marfan syndrome, familial thoracic aortic aneurysms, and sporadic aneurysms.马凡综合征、家族性胸主动脉瘤和散发性动脉瘤患者升主动脉瘤中炎症细胞的特征分析
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引用本文的文献

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Is physical activity a future therapy for patients with Marfan syndrome?体力活动会成为马凡综合征患者的未来疗法吗？
Orphanet J Rare Dis. 2022 Feb 10;17(1):46. doi: 10.1186/s13023-022-02198-9.

本文引用的文献

1
Cardiovascular Benefits of Moderate Exercise Training in Marfan Syndrome: Insights From an Animal Model.马凡综合征患者进行适度运动训练对心血管的益处：动物模型研究的新发现
J Am Heart Assoc. 2017 Sep 25;6(9):e006438. doi: 10.1161/JAHA.117.006438.
2
Aortic microcalcification is associated with elastin fragmentation in Marfan syndrome.主动脉微钙化与马凡综合征中的弹性蛋白断裂有关。
J Pathol. 2017 Nov;243(3):294-306. doi: 10.1002/path.4949. Epub 2017 Sep 21.
3
Mild aerobic exercise blocks elastin fiber fragmentation and aortic dilatation in a mouse model of Marfan syndrome associated aortic aneurysm.
轻度有氧运动可阻止马凡氏综合征相关主动脉瘤小鼠模型中的弹性纤维断裂和主动脉扩张。
J Appl Physiol (1985). 2017 Jul 1;123(1):147-160. doi: 10.1152/japplphysiol.00132.2017. Epub 2017 Apr 6.
4
Nitric oxide mediates aortic disease in mice deficient in the metalloprotease Adamts1 and in a mouse model of Marfan syndrome.一氧化氮介导缺乏金属蛋白酶 Adamts1 的小鼠和马凡综合征小鼠模型中的主动脉疾病。
Nat Med. 2017 Feb;23(2):200-212. doi: 10.1038/nm.4266. Epub 2017 Jan 9.
5
Obesity, Inflammation, and Exercise Training: Relative Contribution of iNOS and eNOS in the Modulation of Vascular Function in the Mouse Aorta.肥胖、炎症与运动训练：诱导型一氧化氮合酶和内皮型一氧化氮合酶在调节小鼠主动脉血管功能中的相对作用
Front Physiol. 2016 Sep 7;7:386. doi: 10.3389/fphys.2016.00386. eCollection 2016.
6
Protective Effect of Irisin on Atherosclerosis via Suppressing Oxidized Low Density Lipoprotein Induced Vascular Inflammation and Endothelial Dysfunction.鸢尾素通过抑制氧化型低密度脂蛋白诱导的血管炎症和内皮功能障碍对动脉粥样硬化的保护作用。
PLoS One. 2016 Jun 29;11(6):e0158038. doi: 10.1371/journal.pone.0158038. eCollection 2016.
7
Exercise does not activate the β3 adrenergic receptor-eNOS pathway, but reduces inducible NOS expression to protect the heart of obese diabetic mice.运动不会激活β3肾上腺素能受体-eNOS通路，但会降低诱导型一氧化氮合酶的表达，从而保护肥胖糖尿病小鼠的心脏。
Basic Res Cardiol. 2016 Jul;111(4):40. doi: 10.1007/s00395-016-0559-0. Epub 2016 May 10.
8
A deleterious gene-by-environment interaction imposed by calcium channel blockers in Marfan syndrome.马凡综合征中钙通道阻滞剂引发的有害基因-环境相互作用。
Elife. 2015 Oct 27;4:e08648. doi: 10.7554/eLife.08648.
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Aortic disease: Losartan versus atenolol in the Marfan aorta-how to treat?主动脉疾病：在马方综合征主动脉病变中氯沙坦与阿替洛尔的比较——如何治疗？
Nat Rev Cardiol. 2015 Aug;12(8):447-8. doi: 10.1038/nrcardio.2015.95. Epub 2015 Jun 16.
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Vascular smooth muscle cell phenotypic changes in patients with Marfan syndrome.马凡综合征患者血管平滑肌细胞的表型变化
Arterioscler Thromb Vasc Biol. 2015 Apr;35(4):960-72. doi: 10.1161/ATVBAHA.114.304412. Epub 2015 Jan 15.