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急性声创伤:如何处理及如何预防?

Acute acoustic trauma: how to manage and how to prevent?

作者信息

Mardassi Ali, Turki Senda, Mbarek Hajer, Hachicha Amani, Benzarti Sonia, Abouda Maher

出版信息

Tunis Med. 2016 Nov;94(11):664.

Abstract

INTRODUCTION

The consequence of an exposure to intense sounds can be a temporary or permanent hearing loss and even with a rapid therapeutic management, severe sensorineural sequelae may persist.

METHODS

the authors report a retrospective study about 64 patients followed for an acute acoustic trauma during a period of 8 years (2006 to 2013). For all the cases, a clinical examination associated to a pure-tone audiometry was conducted. Hearing levels were measured at the frequencies 500, 1000, 2000 and 3000 Hertz. Auditory evoked potentials were performed in 17 cases. The therapeutic and evolutive data were detailed and discussed.

RESULTS

All our patients were male with a mean age of 34 years. The cause of acoustic trauma was a firing of a gun near the ear in 48 cases, an explosion near the ear in 5 cases and a sudden exposure to loud noises near military planes in 11 cases. Clinical complaints were acute hearing loss with tinnitus. Audiometric exams found a sensorineural hearing loss with a hearing level average of 38 decibels (dB) +/- 14 SD. The therapy consisted of systemic cortisteroids associated in all the cases to peripheral vasodilators.  It was given intravenously during 10 days and then orally with vasodilators during one to 3 months. Hyperbaric oxygenotherapy have been administrated for 25 patients. The follow-up consisted of questioning about symptoms, clinical examination and pure-tone audiometry. A good evolution was noted in 52 cases (81%) and the mean of hearing level after therapy was: 24 dB +/- 12 dB. Despite a prolonged therapy with vaso-active drugs, tinnitus persisted in 36 cases.

CONCLUSION

Controlling noise and its harmful effects through technical devices and safety professionals programs are the best way to reduce the frequency and the sensorineural sequelae due to acute acoustic trauma.

摘要

引言

暴露于高强度声音的后果可能是暂时性或永久性听力损失,即使进行快速的治疗处理,严重的感音神经性后遗症仍可能持续存在。

方法

作者报告了一项回顾性研究,该研究对8年间(2006年至2013年)因急性声创伤而接受随访的64例患者进行了分析。对于所有病例,均进行了与纯音听力测定相关的临床检查。在500、1000、2000和3000赫兹频率下测量听力水平。对17例患者进行了听觉诱发电位检查。详细记录并讨论了治疗及病情演变数据。

结果

我们所有的患者均为男性,平均年龄34岁。声创伤的原因包括:48例因在耳边开枪,5例因耳边爆炸,11例因突然暴露于军用飞机附近的巨响。临床症状为急性听力损失伴耳鸣。听力检查发现感音神经性听力损失,平均听力水平为38分贝(dB)±14标准差。治疗方法为在所有病例中均使用全身性皮质类固醇,并联合外周血管扩张剂。静脉给药10天,然后口服血管扩张剂1至3个月。25例患者接受了高压氧治疗。随访包括询问症状、临床检查和纯音听力测定。52例(81%)患者病情好转,治疗后平均听力水平为:24 dB±12 dB。尽管使用血管活性药物进行了长时间治疗,但仍有36例患者耳鸣持续存在。

结论

通过技术设备和安全专业人员计划控制噪声及其有害影响,是减少急性声创伤所致频率及感音神经性后遗症的最佳方法。

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