Commentary: The apolipoprotein A-I mimetic peptide, D-4F, restrains neointimal formation through heme oxygenase-1 up-regulation.
作者信息
Li Volti Giovanni, Avola Roberto, Tibullo Daniele
机构信息
Department of Biomedical and Biotechnological Sciences, University of Catania, Catania, Italy.
出版信息
Front Pharmacol. 2017 Sep 29;8:708. doi: 10.3389/fphar.2017.00708. eCollection 2017.
Abstract
摘要
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Commentary: The apolipoprotein A-I mimetic peptide, D-4F, restrains neointimal formation through heme oxygenase-1 up-regulation.述评：载脂蛋白A-I模拟肽D-4F通过上调血红素加氧酶-1抑制新生内膜形成。
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本文引用的文献
1
The apolipoprotein A-I mimetic peptide, D-4F, restrains neointimal formation through heme oxygenase-1 up-regulation.载脂蛋白 A-I 模拟肽 D-4F 通过上调血红素氧合酶-1 抑制内膜新生。
J Cell Mol Med. 2017 Dec;21(12):3810-3820. doi: 10.1111/jcmm.13290. Epub 2017 Aug 2.
2
The apolipoprotein A-I mimetic peptide, D-4F, alleviates ox-LDL-induced oxidative stress and promotes endothelial repair through the eNOS/HO-1 pathway.载脂蛋白A-I模拟肽D-4F通过eNOS/HO-1途径减轻氧化型低密度脂蛋白诱导的氧化应激并促进内皮修复。
J Mol Cell Cardiol. 2017 Apr;105:77-88. doi: 10.1016/j.yjmcc.2017.01.017. Epub 2017 Mar 6.
3
Apolipoprotein A-I mimetic peptide reverses impaired arterial healing after injury by reducing oxidative stress.载脂蛋白A-I模拟肽通过降低氧化应激反应来逆转损伤后受损的动脉愈合。
Atherosclerosis. 2015 Aug;241(2):709-15. doi: 10.1016/j.atherosclerosis.2015.06.018. Epub 2015 Jun 18.
4
Potential therapeutic effects of natural heme oxygenase-1 inducers in cardiovascular diseases.天然血红素加氧酶-1 诱导剂在心血管疾病中的潜在治疗作用。
Antioxid Redox Signal. 2013 Feb 10;18(5):507-21. doi: 10.1089/ars.2011.4360. Epub 2012 Nov 6.
5
Cholinergic precursors modulate the expression of heme oxigenase-1, p21 during astroglial cell proliferation and differentiation in culture.胆碱能前体在培养的星形胶质细胞增殖和分化过程中调节血红素加氧酶-1、p21 的表达。
Neurochem Res. 2012 Dec;37(12):2795-804. doi: 10.1007/s11064-012-0873-3. Epub 2012 Sep 7.
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Targeting heme oxygenase-1 in vascular disease.靶向血红素加氧酶-1 治疗血管疾病。
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D-4F induces heme oxygenase-1 and extracellular superoxide dismutase, decreases endothelial cell sloughing, and improves vascular reactivity in rat model of diabetes.D-4F可诱导血红素加氧酶-1和细胞外超氧化物歧化酶，减少内皮细胞脱落，并改善糖尿病大鼠模型的血管反应性。
Circulation. 2005 Jun 14;111(23):3126-34. doi: 10.1161/CIRCULATIONAHA.104.517102. Epub 2005 Jun 6.
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