Center for Developmental Science, University of North Carolina at Chapel Hill.
Department of Psychology, Arizona State University.
Dev Psychol. 2018 Feb;54(2):385-396. doi: 10.1037/dev0000419. Epub 2017 Oct 23.
Emerging evidence suggests that family conflict shows continuity across generations and that intergenerational family conflict can be more intense and deleterious than conflict experienced in a single generation. However, few investigations have identified etiological mechanisms by which family conflict is perpetuated across generations. Addressing this limitation, we sampled 246 families from a multigenerational, high-risk, longitudinal study of parents (G1s) and their children (G2s), followed from adolescence to adulthood as well as the children (G3s) of G2 targets. Specifically, the current study examined whether G2s' depressive symptoms measured at multiple time points across development explained continuity in family conflict from 1 generation (G1-G2) to the next (G2-G3). Results revealed that after controlling for externalizing symptoms, depressive symptoms served as mediators of intergenerational family conflict in both men and women, but in different ways. Specifically, G2 women's young adulthood represented a period of vulnerability in which G2 depressive symptoms were especially likely to mediate intergenerational continuity in family conflict. Additionally, in both men and women, higher G1-G2 family conflict was associated with higher depressive symptoms that persisted from adolescence into young adulthood and then subsequently predicted the development of G2-G3 family conflict. Results did not support the hypothesis that G2 partner depressive symptoms moderated the relation between G2 depressive symptoms and G2-G3 family conflict. Implications of findings regarding the roles that G2 gender and G2 depressive symptoms play in the intergenerational transmission of family conflict are discussed. (PsycINFO Database Record
新出现的证据表明,家庭冲突具有代际连续性,并且代际家庭冲突可能比单一代人经历的冲突更强烈和更具危害性。然而,很少有研究确定了家庭冲突在代际间延续的病因机制。为了解决这一局限性,我们从一个多代、高风险、对父母(G1)及其子女(G2)进行纵向研究的多代家庭中抽取了 246 个家庭样本,从青春期一直跟踪到成年期,以及 G2 目标的子女(G3)。具体来说,本研究考察了 G2 在整个发展过程中多次测量的抑郁症状是否可以解释从 1 代(G1-G2)到下一代(G2-G3)的家庭冲突的连续性。结果表明,在控制了外化症状后,抑郁症状在男性和女性中都是代际家庭冲突的中介因素,但方式不同。具体来说,G2 女性的成年早期是一个脆弱时期,在此期间,G2 抑郁症状特别容易介导家庭冲突的代际连续性。此外,在男性和女性中,较高的 G1-G2 家庭冲突与较高的抑郁症状有关,这些症状从青春期持续到成年早期,随后预测了 G2-G3 家庭冲突的发展。结果并不支持 G2 伴侣抑郁症状调节 G2 抑郁症状与 G2-G3 家庭冲突之间关系的假设。讨论了 G2 性别和 G2 抑郁症状在家庭冲突代际传递中所扮演的角色的研究结果的含义。