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Immunoreactivity of ceroid lipofuscin storage pigment in Batten disease with monoclonal antibodies to the amyloid beta-protein.

作者信息

Wisniewski K E, Maslinska D

出版信息

N Engl J Med. 1989 Jan 26;320(4):256-7. doi: 10.1056/nejm198901263200421.

DOI:10.1056/nejm198901263200421
PMID:2911317
Abstract
摘要

相似文献

1
Immunoreactivity of ceroid lipofuscin storage pigment in Batten disease with monoclonal antibodies to the amyloid beta-protein.用针对β-淀粉样蛋白的单克隆抗体检测巴滕病中蜡样脂褐质储存色素的免疫反应性。
N Engl J Med. 1989 Jan 26;320(4):256-7. doi: 10.1056/nejm198901263200421.
2
Beta-protein immunoreactivity in brains of patients with neuronal ceroid lipofuscinosis: ultrastructural and biochemical demonstration.神经元蜡样脂褐质沉积症患者大脑中的β蛋白免疫反应性:超微结构和生化证明
Neurosci Lett. 1990 May 4;112(2-3):155-60. doi: 10.1016/0304-3940(90)90195-f.
3
Topographic heterogeneity of amyloid B-protein epitopes in brains with various forms of neuronal ceroid lipofuscinoses suggesting defective processing of amyloid precursor protein.
Acta Neuropathol. 1990;80(1):26-34. doi: 10.1007/BF00294218.
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Immunoreactivity of neuronal lipofuscin with monoclonal antibodies to the amyloid beta-protein.
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Lipofuscin and ceroid pigments in aging and brain pathology. A review. I. Biochemical and morphological properties.衰老与脑病理学中的脂褐素和类蜡样色素。综述。I. 生化与形态学特性
Rom J Neurol Psychiatry. 1995 Apr-Jun;33(2):121-36.
6
Ceroid, lipofuscin and the ceroid-lipofuscinoses (Batten disease).脂褐素、脂褐质与脂褐质沉积症(巴滕病)
J Inherit Metab Dis. 1993;16(2):280-3. doi: 10.1007/BF00710265.
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Lectin histochemistry in brains with juvenile form of neuronal ceroid-lipofuscinosis (Batten disease).患有青少年型神经元蜡样脂褐质沉积症(巴滕病)的大脑中的凝集素组织化学
Acta Neuropathol. 1990;80(3):274-9. doi: 10.1007/BF00294645.
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You say lipofuscin, we say ceroid: defining autofluorescent storage material.你说脂褐素,我们说类蜡质:定义自发荧光储存物质。
Neurobiol Aging. 2006 Apr;27(4):576-88. doi: 10.1016/j.neurobiolaging.2005.12.006. Epub 2006 Feb 7.
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Sanfilippo disease, type A with some features of ceroid lipofuscinosis.A型桑菲利波病,伴有一些类蜡样脂褐质沉积症的特征。
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Free radicals and aging. Electron spin resonance studies on neuronal lipopigments and cells grown in vitro.
Mech Ageing Dev. 1983 Jul-Aug;22(3-4):335-45. doi: 10.1016/0047-6374(83)90087-8.

引用本文的文献

1
Prominent tauopathy and intracellular β-amyloid accumulation triggered by genetic deletion of cathepsin D: implications for Alzheimer disease pathogenesis.遗传缺失组织蛋白酶 D 引发显著的 tau 病和细胞内 β-淀粉样蛋白积累:对阿尔茨海默病发病机制的影响。
Alzheimers Res Ther. 2024 Apr 4;16(1):70. doi: 10.1186/s13195-024-01443-6.
2
Evidence for lysosomal processing of amyloid beta-protein precursor in cultured cells.培养细胞中淀粉样β蛋白前体溶酶体加工的证据。
Neurochem Res. 1989 Oct;14(10):933-9. doi: 10.1007/BF00965926.
3
Topographic heterogeneity of amyloid B-protein epitopes in brains with various forms of neuronal ceroid lipofuscinoses suggesting defective processing of amyloid precursor protein.
Acta Neuropathol. 1990;80(1):26-34. doi: 10.1007/BF00294218.
4
Enzymatically active lysosomal proteases are associated with amyloid deposits in Alzheimer brain.具有酶活性的溶酶体蛋白酶与阿尔茨海默病大脑中的淀粉样蛋白沉积有关。
Proc Natl Acad Sci U S A. 1990 May;87(10):3861-5. doi: 10.1073/pnas.87.10.3861.
5
Immunohistochemical localization of the proteinase inhibitor region of amyloid precursor proteins in the neocortex of Alzheimer's disease and aged controls.
Acta Neuropathol. 1992;84(3):244-9. doi: 10.1007/BF00227816.