Shiga Sarah A, Werner Frederick W, Garcia-Elias Marc, Harley Brian J
Department of Orthopedic Surgery, SUNY Upstate Medical University, Syracuse, NY.
Department of Orthopedic Surgery, SUNY Upstate Medical University, Syracuse, NY.
J Hand Surg Am. 2018 Apr;43(4):331-338.e2. doi: 10.1016/j.jhsa.2017.10.012. Epub 2017 Nov 14.
To create a biomechanical model of palmar midcarpal instability by selective ligament sectioning and to analyze treatment by simulated partial wrist arthrodesis.
Nine fresh-frozen cadaver arms were moved through 3 servohydraulic actuated motions and 2 passive wrist mobilizations. The dorsal radiocarpal, triquetrohamate, scaphocapitate, and scaphotrapeziotrapezoid ligaments were sectioned to replicate palmar midcarpal instability. Kinematic data for the scaphoid, lunate, and triquetrum were recorded before and after ligament sectioning and again after simulated triquetrohamate arthrodesis (TqHA) and radiolunate arthrodesis (RLA).
Following ligament sectioning, the model we created for palmar midcarpal instability was characterized by significant increases in (1) lunate angular velocity, (2) lunate flexion-extension, and (3) dorsal/volar motion of the capitate during dorsal/volar mobilizations. Simulated TqHA caused significantly more scaphoid flexion and less extension during the wrist radioulnar deviation motion. It also increased the amount of lunate and triquetral extension during wrist flexion-extension. Simulated RLA significantly reduced scaphoid flexion during both wrist radioulnar deviation and flexion-extension.
Both simulated arthrodeses eliminate wrist clunking and may be of value in treating palmar midcarpal instability. However, simulated RLA reduces proximal row motion whereas simulated TqHA alters how the proximal row moves. Long-term clinical studies are needed to determine if these changes are detrimental.
Palmar midcarpal instability is poorly understood, with most treatments based on pathomechanical assumptions. This study provides information that clinicians can use to design better treatment strategies for this unsolved condition.
通过选择性韧带切断术建立掌侧中腕关节不稳定的生物力学模型,并通过模拟部分腕关节融合术分析治疗方法。
对9个新鲜冷冻尸体手臂进行3种伺服液压驱动运动和2种被动腕关节活动。切断背侧桡腕韧带、三角钩骨韧带、舟头韧带和舟大多角小多角韧带,以复制掌侧中腕关节不稳定。在韧带切断术前、后以及模拟三角钩骨融合术(TqHA)和桡月融合术(RLA)后,记录舟骨、月骨和三角骨的运动学数据。
韧带切断术后,我们建立的掌侧中腕关节不稳定模型的特征为:(1)月骨角速度显著增加;(2)月骨屈伸度增加;(3)在背侧/掌侧活动时头状骨的背侧/掌侧运动增加。模拟TqHA在腕关节桡尺偏斜运动时导致舟骨屈曲明显增加,伸展减少。在腕关节屈伸时,它还增加了月骨和三角骨的伸展量。模拟RLA在腕关节桡尺偏斜和屈伸时均显著减少舟骨屈曲。
两种模拟融合术均消除了腕关节弹响,可能对治疗掌侧中腕关节不稳定有价值。然而,模拟RLA减少了近排腕骨的运动,而模拟TqHA改变了近排腕骨的运动方式。需要长期临床研究来确定这些变化是否有害。
掌侧中腕关节不稳定的情况了解较少,大多数治疗基于病理力学假设。本研究提供了临床医生可用于为这种未解决的病症设计更好治疗策略的信息。