Younker D, Meadors C, Coveler L
Department of Anesthesiology, Baylor College of Medicine, Houston 77030.
Chest. 1989 Mar;95(3):687-9. doi: 10.1378/chest.95.3.687.
Several occurrences of pulmonary edema following relief of acute upper airway obstruction have been reported. The edema is associated with normal cardiac filling pressures and responds promptly to conservative therapy. Its origin may be attributed to the cardiopulmonary effects of the vigorous inspiratory effort that the spontaneously breathing patient generates to overcome respiratory obstruction (the Müller maneuver). A patient with postobstruction pulmonary edema complicated by hypovolemia and myocardial infarction is described. Prompt invasive hemodynamic monitoring in selected high-risk patients is suggested.
已有报道称，急性上气道梗阻解除后出现了几例肺水肿。这种水肿与正常的心腔充盈压相关，且对保守治疗反应迅速。其起源可能归因于自主呼吸的患者为克服呼吸阻塞而产生的强烈吸气动作的心肺效应（米勒动作）。本文描述了一名梗阻后肺水肿并发低血容量和心肌梗死的患者。建议对选定的高危患者进行及时的有创血流动力学监测。
