小鼠巨噬细胞、肿瘤细胞与IgG2a同种型抗体之间的破坏性相互作用。 - Suppr | 超能文献

Destructive interactions between murine macrophages, tumor cells, and antibodies of the IgG2a isotype.

作者信息

Johnson W J, Steplewski Z, Koprowski H, Adams D O

出版信息

Adv Exp Med Biol. 1985;184:75-80. doi: 10.1007/978-1-4684-8326-0_6.

DOI:10.1007/978-1-4684-8326-0_6

Abstract

摘要

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Destructive interactions between murine macrophages, tumor cells, and antibodies of the IgG2a isotype.小鼠巨噬细胞、肿瘤细胞与IgG2a同种型抗体之间的破坏性相互作用。

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Hypothesis: macrophages as effector cells for human tumor destruction mediated by monoclonal antibody.假设：巨噬细胞作为单克隆抗体介导的人类肿瘤破坏的效应细胞。

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Mechanisms of tumor growth inhibition.肿瘤生长抑制机制。

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Tumors undergoing rejection induced by monoclonal antibodies of the IgG2a isotype contain increased numbers of macrophages activated for a distinctive form of antibody-dependent cytolysis.由IgG2a同种型单克隆抗体诱导发生排斥反应的肿瘤，含有数量增多的巨噬细胞，这些巨噬细胞被激活以进行一种独特形式的抗体依赖性细胞溶解。

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Simultaneous exposure to FcγR and FcαR on monocytes and macrophages enhances antitumor activity in vivo.单核细胞和巨噬细胞上的FcγR和FcαR同时暴露可增强体内抗肿瘤活性。

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Tumor cell lysis and tumor growth inhibition by the isotype variants of MAb BR55-2 directed against Y oligosaccharide.抗Y寡糖单克隆抗体BR55-2的同型变体对肿瘤细胞的裂解及肿瘤生长的抑制作用

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Antibody-mediated erythrolysis and erythrophagocytosis by human monocytes, macrophages and activated macrophages. Evidence for distinction between involvement of high-affinity and low-affinity receptors for IgG by using different erythroid target cells.抗体介导的人单核细胞、巨噬细胞和活化巨噬细胞的红细胞溶解及红细胞吞噬作用。通过使用不同的红系靶细胞区分IgG高亲和力和低亲和力受体参与情况的证据。

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