出版商更正：精胺合酶缺乏在斯奈德-罗宾逊综合征模型中导致溶酶体功能障碍和氧化应激。
Publisher Correction: Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome.
作者信息
Li Chong, Brazill Jennifer M, Liu Sha, Bello Christofer, Zhu Yi, Morimoto Marie, Cascio Lauren, Pauly Rini, Diaz-Perez Zoraida, Malicdan May Christine V, Wang Hongbo, Boccuto Luigi, Schwartz Charles E, Gahl William A, Boerkoel Cornelius F, Zhai R Grace
机构信息
Department of Molecular and Cellular Pharmacology, University of Miami Miller School of Medicine, 33136, Miami, FL, USA.
School of Pharmacy, Key Laboratory of Molecular Pharmacology and Drug Evaluation (Yantai University), Ministry of Education, Collaborative Innovation Center of Advanced Drug Delivery System and Biotech Drugs in Universities of Shandong, Yantai University, 264005, Yantai, Shandong, China.
出版信息
Nat Commun. 2018 Jan 18;9(1):337. doi: 10.1038/s41467-017-02462-8.
The originally published version of this Article contained errors in Figure 1. In panel c, the grey shading denoting evolutionary conservation and the arrowheads indicating amino acids affected in Snyder-Robinson syndrome were displaced relative to the sequence. These errors have now been corrected in both the PDF and HTML versions of the manuscript.
本文最初发表的版本中图1存在错误。在图c中，表示进化保守性的灰色阴影以及指示在斯奈德-罗宾逊综合征中受影响氨基酸的箭头相对于序列发生了位移。这些错误现已在手稿的PDF和HTML版本中得到更正。
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