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Corrigendum: Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance.

作者信息

Shaffer Sydney M, Dunagin Margaret C, Torborg Stefan R, Torre Eduardo A, Emert Benjamin, Krepler Clemens, Beqiri Marilda, Sproesser Katrin, Brafford Patricia A, Xiao Min, Eggan Elliott, Anastopoulos Ioannis N, Vargas-Garcia Cesar A, Singh Abhyudai, Nathanson Katherine L, Herlyn Meenhard, Raj Arjun

出版信息

Nature. 2018 Mar 8;555(7695):274. doi: 10.1038/nature25162. Epub 2018 Feb 21.

DOI:10.1038/nature25162
PMID:29466340
Abstract

This corrects the article DOI: 10.1038/nature22794.

摘要

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CEACAM1 is a direct SOX10 target and inhibits melanoma immune infiltration and stemness.

本文引用的文献

1
Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance.罕见细胞变异性和药物诱导的重编程作为癌症耐药的一种模式。
Nature. 2017 Jun 15;546(7658):431-435. doi: 10.1038/nature22794. Epub 2017 Jun 7.
癌胚抗原相关细胞黏附分子1（CEACAM1）是SOX10的直接靶点，可抑制黑色素瘤的免疫浸润和干性。
iScience. 2022 Nov 7;25(12):105524. doi: 10.1016/j.isci.2022.105524. eCollection 2022 Dec 22.
4
DNA promoter hypermethylation of melanocyte lineage genes determines melanoma phenotype.黑素细胞谱系基因的 DNA 启动子超甲基化决定了黑色素瘤的表型。
JCI Insight. 2022 Oct 10;7(19):e156577. doi: 10.1172/jci.insight.156577.
5
The State of Melanoma: Emergent Challenges and Opportunities.黑色素瘤现状：新出现的挑战和机遇。
Clin Cancer Res. 2021 May 15;27(10):2678-2697. doi: 10.1158/1078-0432.CCR-20-4092. Epub 2021 Jan 7.