• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

从离体灌注肾中释放组织激肽释放酶。

Release of tissue kallikrein from the isolated perfused kidney.

作者信息

Lâuar N, Bhoola K

出版信息

Adv Exp Med Biol. 1986;198 Pt A:347-54. doi: 10.1007/978-1-4684-5143-6_47.

DOI:10.1007/978-1-4684-5143-6_47
PMID:2949542
Abstract

Experiments were designed to determine the action of regulatory peptides and potassium on the secretion of tissue kallikrein by the isolated perfused rat kidney. Such experiments indicated that in spite of the directly evoked release of kallikrein by arginine-vasopressin (AVP, ADH), oxytocin and potassium from isolated renal cortical slices, the secretion and clearance of active and total tissue kallikrein by the isolated kidney was primarily sensitive to changes in the perfusion pressure.

摘要

设计实验以确定调节肽和钾对离体灌流大鼠肾脏组织激肽释放酶分泌的作用。此类实验表明,尽管精氨酸加压素(AVP,抗利尿激素)、催产素和钾可直接促使离体肾皮质切片释放激肽释放酶,但离体肾脏中活性和总组织激肽释放酶的分泌及清除主要对灌注压力的变化敏感。

相似文献

1
Release of tissue kallikrein from the isolated perfused kidney.从离体灌注肾中释放组织激肽释放酶。
Adv Exp Med Biol. 1986;198 Pt A:347-54. doi: 10.1007/978-1-4684-5143-6_47.
2
Studies on renal and urinary kallikrein in spontaneously hypertensive rats.自发性高血压大鼠肾脏和尿激肽释放酶的研究
Adv Exp Med Biol. 1986;198 Pt B:219-24. doi: 10.1007/978-1-4757-0154-8_27.
3
Decreased renin release and constant kallikrein secretion after injection of L-NAME in isolated perfused rat kidney.在分离灌注的大鼠肾脏中注射L-精氨酸甲酯(L-NAME)后肾素释放减少,激肽释放酶分泌持续。
Life Sci. 1992;50(14):987-93. doi: 10.1016/0024-3205(92)90092-4.
4
Kallikrein release from the kidney: in vitro effects of AVP and DDAVP in three species.肾脏中激肽释放酶的释放:抗利尿激素和去氨加压素对三种物种的体外作用
Arch Int Pharmacodyn Ther. 1988 Mar-Apr;292:281-5.
5
Effects of atrial natriuretic peptides on metabolism of arginine vasopressin by isolated perfused rat kidney.心房利钠肽对离体灌注大鼠肾脏精氨酸加压素代谢的影响。
Am J Physiol. 1992 Aug;263(2 Pt 2):R273-8. doi: 10.1152/ajpregu.1992.263.2.R273.
6
Effect of changes in perfusion pressure on renal and urinary kallikrein in the isolated perfused rat kidney.灌注压力变化对离体灌注大鼠肾脏中肾及尿激肽释放酶的影响。
Ren Physiol. 1983;6(6):288-94. doi: 10.1159/000172914.
7
Release of active and inactive kallikrein from the isolated perfused rat kidney.从离体灌注大鼠肾脏中释放活性和非活性激肽释放酶。
Clin Sci (Lond). 1984 Feb;66(2):207-15. doi: 10.1042/cs0660207.
8
Vasopressin stimulates urinary kallikrein excretion in the isolated erythrocyte-perfused rat kidney.血管加压素可刺激离体红细胞灌注大鼠肾脏中尿激肽释放酶的排泄。
Ren Physiol Biochem. 1988 Jan-Feb;11(1-2):50-9. doi: 10.1159/000173149.
9
[Kallikrein secretion by the isolated perfused rat kidney. Role of perfusion pressure and the renin-angiotensin system].[离体灌注大鼠肾脏的激肽释放酶分泌。灌注压力和肾素-血管紧张素系统的作用]
Arch Mal Coeur Vaiss. 1985 Oct;78(11):1677-80.
10
Polarity of kallikrein release and activation in perfused rat kidneys.灌注大鼠肾脏中激肽释放酶释放与激活的极性
Am J Physiol. 1990 Nov;259(5 Pt 2):F752-7. doi: 10.1152/ajprenal.1990.259.5.F752.

引用本文的文献

1
Angiotensin II type 2 receptor overexpression activates the vascular kinin system and causes vasodilation.血管紧张素II 2型受体过表达激活血管激肽系统并导致血管舒张。
J Clin Invest. 1999 Oct;104(7):925-35. doi: 10.1172/JCI7886.
2
A tissue kallikrein in the synovial fluid of patients with rheumatoid arthritis.类风湿性关节炎患者滑液中的一种组织激肽释放酶。
Ann Rheum Dis. 1989 Feb;48(2):128-33. doi: 10.1136/ard.48.2.128.