多巴胺D2受体刺激可降低大鼠纹状体切片的肌醇三磷酸水平。 - Suppr

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超能文献

Dopamine D2 receptor stimulation decreases the inositol trisphosphate level of rat striatal slices.

作者信息

Pizzi M, D'Agostini F, Da Prada M, Spano P F, Haefely W E

出版信息

Eur J Pharmacol. 1987 Apr 14;136(2):263-4. doi: 10.1016/0014-2999(87)90724-2.

DOI:10.1016/0014-2999(87)90724-2

PMID:2954833

Abstract

摘要

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Dopamine D2 receptor stimulation decreases the inositol trisphosphate level of rat striatal slices.多巴胺D2受体刺激可降低大鼠纹状体切片的肌醇三磷酸水平。

Eur J Pharmacol. 1987 Apr 14;136(2):263-4. doi: 10.1016/0014-2999(87)90724-2.

Dopamine D2 receptor stimulation inhibits inositol phosphate generating system in rat striatal slices.

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The effect of dopamine D1 and D2 receptor agonists on inositol phosphate turnover in rat striatal slices.多巴胺D1和D2受体激动剂对大鼠纹状体切片中肌醇磷酸周转率的影响。

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Further evidence against the coupling of dopamine receptors to phosphoinositide hydrolysis in rat striatum.

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Dopamine may influence striatal GABA release via three separate mechanisms.多巴胺可能通过三种不同机制影响纹状体γ-氨基丁酸（GABA）的释放。

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Sustained activation does not desensitize the dopamine D2 receptor-mediated control of evoked in vitro release of radiolabeled acetylcholine from rat striatum.

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D1 dopamine receptor stimulation enables the postsynaptic, but not autoreceptor, effects of D2 dopamine agonists in nigrostriatal and mesoaccumbens dopamine systems.D1多巴胺受体刺激可使黑质纹状体和中伏隔核多巴胺系统中D2多巴胺激动剂产生突触后效应，但不产生自身受体效应。

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The D-2 agonist quinpirole releases striatal dopamine in vivo.D2激动剂喹吡罗在体内释放纹状体多巴胺。

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The glycosylation properties of D2 dopamine receptors from striatal and limbic areas of bovine brain.牛脑纹状体和边缘区域D2多巴胺受体的糖基化特性

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Association of two pertussis toxin-sensitive G-proteins with the D2-dopamine receptor from bovine striatum.两种百日咳毒素敏感的G蛋白与牛纹状体D2 - 多巴胺受体的关联。

EMBO J. 1989 Aug;8(8):2353-7. doi: 10.1002/j.1460-2075.1989.tb08363.x.

Neonatal dopamine lesion in the rat results in enhanced adenylate cyclase activity without altering dopamine receptor binding or dopamine- and adenosine 3':5'-monophosphate-regulated phosphoprotein (DARPP-32) immunoreactivity.大鼠新生期多巴胺损伤导致腺苷酸环化酶活性增强，而不改变多巴胺受体结合或多巴胺和3':5'-环磷酸腺苷调节磷蛋白（DARPP-32）免疫反应性。

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