心肌蛋白合成的缺氧抑制研究。
Studies on the anoxic inhibition of myocardial protein synthesis.
作者信息
Lesch M, Peterson M B
出版信息
Recent Adv Stud Cardiac Struct Metab. 1975;8:101-15.
PMID:2956
Abstract
The present study demonstrates that exposure of cardiac muscle to high levels of glucose during anoxia appears to retard damage to myocardial protein synthesis. The mechanism of this "glucose" effect is glucose-specific and appears related to the intracellular metabolism of glucose by the anoxic myocardium.
摘要
本研究表明，在缺氧期间将心肌暴露于高水平葡萄糖下似乎会延缓对心肌蛋白质合成的损伤。这种“葡萄糖”效应的机制具有葡萄糖特异性，并且似乎与缺氧心肌对葡萄糖的细胞内代谢有关。
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