Jacobson D M, Warner J J, Choucair A K, Ptacek L J
Department of Neurology, Marshfield Clinic, Wisconsin 54449.
J Clin Neuroophthalmol. 1988 Dec;8(4):263-8.
Trauma-induced superior oblique palsy usually results from contusion or avulsion of the trochlear nerve or from decompensation of a congenital trochlear nerve palsy. Severe craniocerebral trauma is often associated with the former mechanism, whereas more minor closed-head injuries can decompensate a congenital phoria. We report a patient who developed an isolated trochlear nerve palsy following minor head trauma. Investigation revealed an unsuspected tentorial vascular malformation that was compressing the trochlear nerve in its subarachnoid course. In the absence of other features (e.g., documentation of old head tilt, large vertical fusion amplitudes) that support decompensation of a congenital phoria, compressive lesions should be sought in cases of fourth cranial nerve palsies that follow minor head trauma.
创伤性上斜肌麻痹通常由滑车神经挫伤或撕脱,或先天性滑车神经麻痹失代偿引起。严重颅脑创伤常与前一种机制相关,而较轻的闭合性头部损伤可导致先天性隐斜失代偿。我们报告1例轻度头部创伤后发生孤立性滑车神经麻痹的患者。检查发现一个意外的小脑幕血管畸形,在蛛网膜下腔行程中压迫滑车神经。在没有其他支持先天性隐斜失代偿的特征(如既往头部倾斜记录、大的垂直融合幅度)的情况下,对于轻度头部创伤后出现的动眼神经麻痹病例,应寻找压迫性病变。