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低频电磁场可减少人源心肌细胞缺血再灌注损伤,并支持其代谢功能。

Low frequency electromagnetic field decreases ischemia-reperfusion injury of human cardiomyocytes and supports their metabolic function.

机构信息

1 Department and Clinic of Cardiology, Wroclaw Medical University, Wroclaw 50-556, Poland.

2 Department and Clinic of Emergency Medicine, Wroclaw Medical University, Wroclaw 50-556, Poland.

出版信息

Exp Biol Med (Maywood). 2018 Jun;243(10):809-816. doi: 10.1177/1535370218779773. Epub 2018 May 30.

Abstract

Electromagnetic field at extremely low frequencies plays a significant role in the physiological function of human tissues and systems. It is shown that electromagnetic field inhibits production of reactive oxygen species which are involved in heart injury triggered by oxidative stress. We hypothesize that low frequency electromagnetic field protects function of cardiac cells from ischemia-reperfusion injury. Human cardiac myocytes, endothelial cells, and cardiac fibroblast underwent ischemia-reperfusion conditions in the presence or in the absence of low frequency electromagnetic field. LDH and MMP-2 activities (as markers of cell injury), and cell metabolic activity (by fluorescein diacetate staining) were measured to determine the protective role of low frequency electromagnetic field. Our data showed that short courses of low frequency electromagnetic field protect cardiac cells from cellular damage and preserve their metabolic activity during ischemia-reperfusion. This study demonstrates the possibility to use of low frequency electromagnetic field as strategy for the prevention or therapy of ischemia-reperfusion injury. Impact statement In our study, we showed that LF-EMF may be protective for heart during ischemia-reperfusion (I/R). Following is the short description of the main findings: (a) the response to the I/R injury was different for endothelial cells, fibroblasts, and cardiomyocytes; (b) I/R decreases MMP-2 activity in cardiac myocytes and fibroblasts;

摘要

极低频电磁场在人体组织和系统的生理功能中发挥着重要作用。研究表明,电磁场可以抑制活性氧的产生,而活性氧参与了氧化应激引起的心脏损伤。我们假设低频电磁场可以保护心脏细胞免受缺血再灌注损伤。在存在或不存在低频电磁场的情况下,让人心肌细胞、内皮细胞和心肌成纤维细胞经历缺血再灌注条件。通过测量乳酸脱氢酶(LDH)和基质金属蛋白酶-2(MMP-2)的活性(作为细胞损伤的标志物)以及细胞代谢活性(通过荧光素二乙酸酯染色),来确定低频电磁场的保护作用。我们的数据表明,短暂的低频电磁场可以保护心脏细胞免受细胞损伤,并在缺血再灌注期间保持其代谢活性。这项研究表明,低频电磁场作为预防或治疗缺血再灌注损伤的策略具有可能性。影响说明在我们的研究中,我们表明 LF-EMF 可能对缺血再灌注(I/R)期间的心脏具有保护作用。以下是主要发现的简短描述:(a)I/R 对内皮细胞、成纤维细胞和心肌细胞的反应不同;(b)I/R 降低了心肌细胞和成纤维细胞中 MMP-2 的活性;

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