• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

相似文献

1
A Phenome-Wide Association Study Uncovers a Role for Autoimmunity in the Development of Chronic Obstructive Pulmonary Disease.一项全表型关联研究揭示自身免疫在慢性阻塞性肺疾病发生中的作用。
Am J Respir Cell Mol Biol. 2018 Jun;58(6):777-779. doi: 10.1165/rcmb.2017-0409LE.
2
Common genetic variants associated with resting oxygenation in chronic obstructive pulmonary disease.与慢性阻塞性肺疾病静息氧合相关的常见遗传变异。
Am J Respir Cell Mol Biol. 2014 Nov;51(5):678-87. doi: 10.1165/rcmb.2014-0135OC.
3
Network Analysis of Genome-Wide Association Studies for Chronic Obstructive Pulmonary Disease in the Context of Biological Pathways.在生物通路背景下慢性阻塞性肺疾病全基因组关联研究的网络分析
Am J Respir Crit Care Med. 2019 Dec 1;200(11):1439-1441. doi: 10.1164/rccm.201904-0902LE.
4
Editorial: Proteostasis-imbalance and pathogenesis of chronic obstructive lung diseases.
Curr Mol Med. 2012 Aug;12(7):805-6. doi: 10.2174/156652412801318737.
5
Visual Assessment of Chest Computed Tomographic Images Is Independently Useful for Genetic Association Analysis in Studies of Chronic Obstructive Pulmonary Disease.胸部计算机断层扫描图像的视觉评估可独立用于慢性阻塞性肺疾病研究中的遗传关联分析。
Ann Am Thorac Soc. 2017 Jan;14(1):33-40. doi: 10.1513/AnnalsATS.201606-427OC.
6
CTLA4 polymorphisms and COPD.细胞毒性T淋巴细胞相关抗原4(CTLA4)基因多态性与慢性阻塞性肺疾病(COPD)
Eur Respir J. 2010 Feb;35(2):457-8. doi: 10.1183/09031936.00134709.
7
[Autoimmunity in pathogenesis of chronic obstructive pulmonary disease].[自身免疫在慢性阻塞性肺疾病发病机制中的作用]
Medicina (Kaunas). 2005;41(3):190-5.
8
CTLA-4 gene polymorphisms and susceptibility to chronic obstructive pulmonary disease.细胞毒性T淋巴细胞相关抗原4(CTLA-4)基因多态性与慢性阻塞性肺疾病易感性
Int J Clin Exp Pathol. 2013 Oct 15;6(11):2548-53. eCollection 2013.
9
Autoimmunity: lungs and citrullination.自身免疫:肺部与瓜氨酸化。
Nat Rev Rheumatol. 2015 May;11(5):261-2. doi: 10.1038/nrrheum.2015.38. Epub 2015 Mar 17.
10
Genetic polymorphism and chronic obstructive pulmonary disease.基因多态性与慢性阻塞性肺疾病
Int J Chron Obstruct Pulmon Dis. 2017 May 10;12:1385-1393. doi: 10.2147/COPD.S134161. eCollection 2017.

引用本文的文献

1
Is There a Link between Chronic Obstructive Pulmonary Disease and Lung Adenocarcinoma? A Clinico-Pathological and Molecular Study.慢性阻塞性肺疾病与肺腺癌之间存在关联吗?一项临床病理与分子研究。
J Pers Med. 2024 Aug 8;14(8):839. doi: 10.3390/jpm14080839.
2
Circadian clock-based therapeutics in chronic pulmonary diseases.基于生物钟的慢性肺部疾病治疗方法。
Trends Pharmacol Sci. 2022 Dec;43(12):1014-1029. doi: 10.1016/j.tips.2022.09.004. Epub 2022 Oct 24.
3
Dissociation between airway and systemic autoantibody responses in chronic obstructive pulmonary disease.慢性阻塞性肺疾病中气道与全身自身抗体反应的分离
Ann Transl Med. 2020 Aug;8(15):918. doi: 10.21037/atm-20-944.
4
Macrophages-the immune effector guardians of the lung: impact of corticosteroids on their functional responses.巨噬细胞——肺部的免疫效应卫士:皮质类固醇对其功能反应的影响。
Clin Sci (Lond). 2020 Jul 17;134(13):1631-1635. doi: 10.1042/CS20200382.
5
Obstructive lung diseases and risk of rheumatoid arthritis.阻塞性肺疾病与类风湿关节炎风险。
Expert Rev Clin Immunol. 2020 Jan;16(1):37-50. doi: 10.1080/1744666X.2019.1698293. Epub 2020 Jan 6.

本文引用的文献

1
Unravelling the human genome-phenome relationship using phenome-wide association studies.利用表型全基因组关联研究揭示人类基因组-表型关系。
Nat Rev Genet. 2016 Mar;17(3):129-45. doi: 10.1038/nrg.2015.36. Epub 2016 Feb 15.
2
Autoreactivity to glucose regulated protein 78 links emphysema and osteoporosis in smokers.对葡萄糖调节蛋白78的自身反应性将吸烟者的肺气肿和骨质疏松症联系起来。
PLoS One. 2014 Sep 12;9(9):e105066. doi: 10.1371/journal.pone.0105066. eCollection 2014.
3
The NHGRI GWAS Catalog, a curated resource of SNP-trait associations.NHGRI GWAS Catalog,一个经过精心策划的 SNP 与特征关联资源。
Nucleic Acids Res. 2014 Jan;42(Database issue):D1001-6. doi: 10.1093/nar/gkt1229. Epub 2013 Dec 6.
4
Systematic comparison of phenome-wide association study of electronic medical record data and genome-wide association study data.系统比较电子病历数据的表型全基因组关联研究和全基因组关联研究数据。
Nat Biotechnol. 2013 Dec;31(12):1102-10. doi: 10.1038/nbt.2749.
5
Excessive Th1 responses due to the absence of TGF-β signaling cause autoimmune diabetes and dysregulated Treg cell homeostasis.由于 TGF-β 信号缺失导致的过度 Th1 反应会引起自身免疫性糖尿病和调节性 T 细胞稳态失调。
Proc Natl Acad Sci U S A. 2013 Apr 23;110(17):6961-6. doi: 10.1073/pnas.1304498110. Epub 2013 Apr 8.
6
Genome-wide association analysis of blood biomarkers in chronic obstructive pulmonary disease.全基因组关联分析在慢性阻塞性肺疾病血液生物标志物中的应用。
Am J Respir Crit Care Med. 2012 Dec 15;186(12):1238-47. doi: 10.1164/rccm.201206-1013OC. Epub 2012 Nov 9.
7
Autoimmunity in chronic obstructive pulmonary disease: clinical and experimental evidence.慢性阻塞性肺疾病中的自身免疫:临床和实验证据。
Expert Rev Clin Immunol. 2012 Mar;8(3):285-92. doi: 10.1586/eci.12.7.
8
A genome-wide association study suggests contrasting associations in ACPA-positive versus ACPA-negative rheumatoid arthritis.一项全基因组关联研究表明,在 ACPA 阳性和 ACPA 阴性类风湿关节炎中存在相反的关联。
Ann Rheum Dis. 2011 Feb;70(2):259-65. doi: 10.1136/ard.2009.126821. Epub 2010 Dec 14.
9
Genome-wide association study meta-analysis identifies seven new rheumatoid arthritis risk loci.全基因组关联研究荟萃分析确定了七个新的类风湿关节炎风险位点。
Nat Genet. 2010 Jun;42(6):508-14. doi: 10.1038/ng.582. Epub 2010 May 9.
10
Identification of genes and haplotypes that predict rheumatoid arthritis using random forests.使用随机森林识别预测类风湿性关节炎的基因和单倍型。
BMC Proc. 2009 Dec 15;3 Suppl 7(Suppl 7):S68. doi: 10.1186/1753-6561-3-s7-s68.

A Phenome-Wide Association Study Uncovers a Role for Autoimmunity in the Development of Chronic Obstructive Pulmonary Disease.

作者信息

Ji Xiangming, Niu Xinnan, Qian Jun, Martucci Victoria, Pendergrass Sarah A, Gorlov Ivan P, Amos Christopher I, Denny Joshua C, Massion Pierre P, Aldrich Melinda C

机构信息

2 Georgia State University Atlanta, Georgia.

1 Vanderbilt University Medical Center Nashville, Tennessee.

出版信息

Am J Respir Cell Mol Biol. 2018 Jun;58(6):777-779. doi: 10.1165/rcmb.2017-0409LE.

DOI:10.1165/rcmb.2017-0409LE
PMID:29856256
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6002656/
Abstract
摘要