College of Nursing and Health Innovation and.
Department of Kinesiology, University of Texas at Arlington, Arlington, TX.
Exerc Sport Sci Rev. 2018 Jul;46(3):144-151. doi: 10.1249/JES.0000000000000149.
We hypothesize that the reduced peak aerobic power (peak V˙O2) after heart transplantation is due to impaired cardiovascular and skeletal muscle function, and its improvement with short-term (≤1 yr) exercise training is primarily due to favorable skeletal muscle adaptations. Furthermore, the increased peak V˙O2 with long-term (>2 yr) training is primarily mediated by cardiac (sympathetic) reinnervation.
我们假设,心脏移植后峰值有氧能力(峰值 VO2)降低是由于心血管和骨骼肌功能受损所致,而短期(≤1 年)运动训练后的改善主要归因于骨骼肌的适应性改变。此外,长期(>2 年)训练后峰值 VO2 的增加主要是通过心脏(交感)神经再支配来介导的。