Kerasnoudis A, Ntasiou P, Barmpalios G
Dept. of Neurology, St. Luke's Hospital, Thessaloniki, Greece; Dept. of Neurology, St. Josef Hospital, Bochum, Germany.
Dept. of Neurology, St. Luke's Hospital, Thessaloniki, Greece.
J Electromyogr Kinesiol. 2018 Oct;42:20-23. doi: 10.1016/j.jelekin.2018.06.004. Epub 2018 Jun 13.
We report on the prognostic role of cross sectional area (CSA) enlargement and axonal damage in traumatic sural neuropathy (TSN).
Reference values were defined in 23 healthy subjects. 13patients with TSN underwent evaluation (Thessaloniki Hypesthesia Score (THS), ultrasound, electrophysiology). All patients were followed up with THS 6 months after initial evaluation.
During initial evaluation, the 13 patients showed a mean THS of 2.6 (SD ± 0.9). 7 patients showed pathological (pUS) and 6 normal CSA (nUS). 8 patients showed axonal affection (pCS) and 5 no axonal affection (nCS). During follow up, mean THS was 3.1 (SD ± 0.9) in pUS, and 1.8 (SD ± 0.7) in the nUS group (p < 0.001). Mean THS was 2.8 (SD ± 0.7) in pCS, and 2.1 (SD ± 0.9) in nCS group (p = 0.035).
CSA enlargement, but not axonal loss, seems to have a negative prognostic role in patients with TSN.
我们报告了横截面积(CSA)增大和轴突损伤在创伤性腓肠神经病变(TSN)中的预后作用。
定义了23名健康受试者的参考值。13例TSN患者接受了评估(塞萨洛尼基感觉减退评分(THS)、超声、电生理学)。所有患者在初次评估后6个月采用THS进行随访。
在初次评估期间,13例患者的平均THS为2.6(标准差±0.9)。7例患者显示CSA病理性改变(pUS),6例患者CSA正常(nUS)。8例患者显示轴突受累(pCS),5例患者无轴突受累(nCS)。随访期间,pUS组的平均THS为3.1(标准差±0.9),nUS组为1.8(标准差±0.7)(p<0.001)。pCS组的平均THS为2.8(标准差±0.7),nCS组为2.1(标准差±0.9)(p=0.035)。
CSA增大而非轴突丢失似乎在TSN患者中具有不良预后作用。