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Genetic Risk in Coronary Artery Disease.冠状动脉疾病中的遗传风险。
Arq Bras Cardiol. 2018 Jul;111(1):62-63. doi: 10.5935/abc.20180130.
2
Family or SNPs: what counts for hereditary risk of coronary artery disease?
Eur Heart J. 2016 Feb 7;37(6):568-71. doi: 10.1093/eurheartj/ehv545. Epub 2015 Oct 15.
3
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4
Genetic variation in vitamin D receptor gene (Fok1:rs2228570) is associated with risk of coronary artery disease.维生素D受体基因（Fok1:rs2228570）的基因变异与冠状动脉疾病风险相关。
Biomarkers. 2017 May-Jun;22(3-4):387. doi: 10.1080/1354750X.2016.1204008. Epub 2016 Jul 28.
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Expression of the Marburg I Single Nucleotide Polymorphism (MI-SNP) and the Marburg II Single Nucleotide Polymorphism (MII-SNP) of the Factor VII-Activating Protease (FSAP) Gene and Risk of Coronary Artery Disease (CAD): A Pilot Study in a Single Population.载脂蛋白 M 单核苷酸多态性（apoM-SNP）和载脂蛋白 A-V 单核苷酸多态性（apoA-V-SNP）与冠状动脉疾病（CAD）的相关性：一项单人群初步研究。
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Functional Analysis of a Novel Genome-Wide Association Study Signal in SMAD3 That Confers Protection From Coronary Artery Disease.SMAD3中一个新的全基因组关联研究信号的功能分析，该信号赋予对冠状动脉疾病的保护作用。
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Complex disease: From non-coding risk variant to biological mechanism in CAD.
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Socioeconomic Status Interacts with the Genetic Effect of a Chromosome 9p21.3 Common Variant to Influence Coronary Artery Calcification and Incident Coronary Events in the Heinz Nixdorf Recall Study (Risk Factors, Evaluation of Coronary Calcium, and Lifestyle).在海因茨·尼克斯多夫召回研究（风险因素、冠状动脉钙化评估和生活方式）中，社会经济地位与9号染色体p21.3常见变异的遗传效应相互作用，影响冠状动脉钙化和冠心病事件。
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[Association of single nucleotide polymorphism on chromosome 12q24.31 with susceptibility to coronary artery disease].12号染色体q24.31区域单核苷酸多态性与冠状动脉疾病易感性的关联
Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2011 Aug;28(4):455-9. doi: 10.3760/cma.j.issn.1003-9406.2011.04.022.

引用本文的文献

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Influence of genetic polymorphisms on serum biomarkers of cardiac health.遗传多态性对心脏健康血清生物标志物的影响。
Medicine (Baltimore). 2023 Jun 9;102(23):e33953. doi: 10.1097/MD.0000000000033953.
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Magnesium Supplementation Affects the Expression of Sirtuin1, Tumor Protein P53 and Endothelial Nitric Oxide Synthase Genes in Patients with Atherosclerosis: A Double-Blind, Randomized, Placebo-Controlled Trial.补充镁对动脉粥样硬化患者沉默调节蛋白1、肿瘤蛋白P53和内皮型一氧化氮合酶基因表达的影响：一项双盲、随机、安慰剂对照试验
Indian J Clin Biochem. 2023 Jan;38(1):59-66. doi: 10.1007/s12291-022-01032-0. Epub 2022 Apr 9.
3
Vitamin D-binding protein and vitamin D receptor genotypes and 25-hydroxyvitamin D levels are associated with development of aortic and mitral valve calcification and coronary artery diseases.维生素 D 结合蛋白和维生素 D 受体基因型与 25-羟维生素 D 水平与主动脉瓣和二尖瓣钙化及冠状动脉疾病的发生有关。
Mol Biol Rep. 2019 Oct;46(5):5225-5236. doi: 10.1007/s11033-019-04979-1. Epub 2019 Jul 29.

本文引用的文献

1
Genetic Risk Analysis of Coronary Artery Disease in a Population-based Study in Portugal, Using a Genetic Risk Score of 31 Variants.在葡萄牙一项基于人群的研究中，利用31个变异体的遗传风险评分对冠状动脉疾病进行遗传风险分析。
Arq Bras Cardiol. 2018 Jul;111(1):50-61. doi: 10.5935/abc.20180107. Epub 2018 Jul 2.
2
A decade of genome-wide association studies for coronary artery disease: the challenges ahead.一项针对冠心病的全基因组关联研究十年：未来的挑战。
Cardiovasc Res. 2018 Jul 15;114(9):1241-1257. doi: 10.1093/cvr/cvy084.
3
Prospective association of a genetic risk score with major adverse cardiovascular events in patients with coronary artery disease.冠心病患者基因风险评分与主要不良心血管事件的前瞻性关联
Medicine (Baltimore). 2017 Dec;96(51):e9473. doi: 10.1097/MD.0000000000009473.
4
Genetic polymorphisms offer insight into the causal role of microRNA in coronary artery disease.遗传多态性为 microRNA 在冠心病中的因果作用提供了深入了解。
Atherosclerosis. 2018 Feb;269:63-70. doi: 10.1016/j.atherosclerosis.2017.12.022. Epub 2017 Dec 15.
5
Genome-Wide Linkage Analysis of Large Multiple Multigenerational Families Identifies Novel Genetic Loci for Coronary Artery Disease.全基因组连锁分析大型多代家族，鉴定冠状动脉疾病新的遗传位点。
Sci Rep. 2017 Jul 14;7(1):5472. doi: 10.1038/s41598-017-05381-2.
6
Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms.15个新的冠状动脉疾病风险位点突显动脉壁特异性机制。
Nat Genet. 2017 Jul;49(7):1113-1119. doi: 10.1038/ng.3874. Epub 2017 May 22.
7
Identification of the Functional Variant(s) that Explain the Low-Density Lipoprotein Receptor (LDLR) GWAS SNP rs6511720 Association with Lower LDL-C and Risk of CHD.鉴定解释低密度脂蛋白受体（LDLR）全基因组关联研究单核苷酸多态性（SNP）rs6511720与较低低密度脂蛋白胆固醇（LDL-C）及冠心病风险之间关联的功能变异。
PLoS One. 2016 Dec 14;11(12):e0167676. doi: 10.1371/journal.pone.0167676. eCollection 2016.
8
Genetics of Coronary Artery Disease.冠状动脉疾病的遗传学。
Circ Res. 2016 Feb 19;118(4):564-78. doi: 10.1161/CIRCRESAHA.115.306566.
9
Identifying Novel Gene Variants in Coronary Artery Disease and Shared Genes With Several Cardiovascular Risk Factors.识别冠状动脉疾病中的新型基因变异以及与多种心血管危险因素相关的共享基因。
Circ Res. 2016 Jan 8;118(1):83-94. doi: 10.1161/CIRCRESAHA.115.306629. Epub 2015 Oct 20.
10
Genome-wide significant loci: how important are they? Systems genetics to understand heritability of coronary artery disease and other common complex disorders.全基因组显著位点：它们有多重要？利用系统遗传学理解冠状动脉疾病及其他常见复杂疾病的遗传力。
J Am Coll Cardiol. 2015 Mar 3;65(8):830-845. doi: 10.1016/j.jacc.2014.12.033.

Genetic Risk in Coronary Artery Disease.

作者信息

Martinez Paula F, Okoshi Marina P

机构信息

Faculdade de Fisioterapia da Universidade de Mato Grosso do Sul, Campo Grande, MS - Brazil.

Faculdade de Medicina de Botucatu (UNESP), Botucatu, SP - Brazil.

出版信息

Arq Bras Cardiol. 2018 Jul;111(1):62-63. doi: 10.5935/abc.20180130.

DOI:10.5935/abc.20180130
PMID:30110045
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6078374/
Abstract
摘要