Al Namat Razan, Mitu O, Ghiciuc Cristina, Felea Maura, Mitu F
Rev Med Chir Soc Med Nat Iasi. 2016 Oct-Dec;120(4):850-4.
Antiphospholipid syndrome (APS), one of the most common states of acquired hypercoagulability, is diagnosed by the persistent presence of antiphospholipid antibodies and recurrent episodes of vascular thrombosis. We present the case of a 39-year-old man late-presenting for cardiac rehabilitation treatment after primary percutaneous coronary intervention (PCI) performed for anteroseptal myocardial infarction. He was a nonsmoker, with no prior personal history of other cardiovascular diseases (CVD) or cardiometabolic syndrome. The 60% thrombotic occlusion of the left anterior descending artery (LAD) leading to the acute cardiac event was the only abnormality that was found. The only etiological explanation was the late measurement and the positive tests for antiphospholipid antibodies. In young patients with no history of thrombotic disorder, such as cancer, cardiovascular or metabolic diseases, the unexpected onset of myocardial infarction by thrombotic coronary occlusion can be attributed to silent, undiagnosed autoimmune condition.
抗磷脂综合征（APS）是获得性高凝状态最常见的情况之一，通过抗磷脂抗体的持续存在和血管血栓形成的反复发作来诊断。我们报告了一例39岁男性患者，在因前间隔心肌梗死接受初次经皮冠状动脉介入治疗（PCI）后延迟接受心脏康复治疗。他不吸烟，既往无其他心血管疾病（CVD）或心脏代谢综合征个人史。导致急性心脏事件的左前降支（LAD）60%血栓性闭塞是唯一发现的异常。唯一的病因解释是抗磷脂抗体检测较晚且结果呈阳性。在没有血栓形成性疾病病史（如癌症、心血管或代谢疾病）的年轻患者中，血栓性冠状动脉闭塞导致的意外心肌梗死发作可归因于隐匿的、未被诊断的自身免疫性疾病。
