Yamamoto Masaki, Tashiro Miwa, Noguchi Tatsuya, Orihashi Kazumasa
Department of Operations Management, Kochi Medical School, Kochi University, Kochi, Japan.
Department of Surgery, Kochi Medical School, Kochi University, Kochi, Japan.
J Cardiol Cases. 2018 May 31;18(3):92-94. doi: 10.1016/j.jccase.2018.05.005. eCollection 2018 Sep.
A 69-year-old woman with a history of graft replacement for abdominal aortic aneurysm developed a complicated type B aortic dissection that resulted in renal malperfusion after thoracic endovascular aortic repair (TEVAR). The primary entry tear was formed at the aortic arch and the false lumen (FL) continued to the abdominal aorta. The distal end of the FL formed a pouch-like blind alley above the suture line of the bifurcated abdominal graft. The true lumen (TL) was compressed and caused severe limb ischemia. The right renal artery (rRA) originated from the FL. The patient had initially undergone emergent axillofemoral bypass for limb malperfusion. Three weeks later, restenosis of the TL caused visceral malperfusion, and a huge thrombus formed in the FL pouch. TEVAR expanded the TL and restored visceral vascularization. However, the expanded TL compressed the thrombus in the blind alley to the rRA orifice and caused right renal malperfusion. The history of abdominal graft replacement may have increased the risk of a thrombotic event after TEVAR. Primary TEVAR may have helped prevent thrombus formation in the FL pouch. < This case report describes visceral arterial embolism induced by endovascular aortic repair (TEVAR) for type B aortic dissection in a patient with prior abdominal aortic grafting. A huge thrombus formed in the blind pouch of the false lumen (FL) above the graft suture line. TEVAR expanded the true lumen and the FL thrombus occluded a visceral artery with FL origin. Post-graft replacement has a risk of thrombus formation in the FL pouch and thrombus compression in visceral arteries.>.
一名有腹主动脉瘤移植置换史的69岁女性,在胸主动脉腔内修复术(TEVAR)后发生了复杂的B型主动脉夹层,并导致肾灌注不良。原发性入口撕裂位于主动脉弓,假腔(FL)延伸至腹主动脉。FL的远端在分叉腹主动脉移植物缝线处上方形成袋状死胡同。真腔(TL)受压,导致严重的肢体缺血。右肾动脉(rRA)起源于FL。患者最初因肢体灌注不良接受了急诊腋股旁路手术。三周后,TL再狭窄导致内脏灌注不良,FL袋内形成巨大血栓。TEVAR扩张了TL并恢复了内脏血管化。然而,扩张的TL将死胡同内的血栓挤压到rRA开口处,导致右肾灌注不良。腹主动脉移植置换史可能增加了TEVAR后血栓形成的风险。初次TEVAR可能有助于预防FL袋内血栓形成。<本病例报告描述了在一名既往有腹主动脉移植史的患者中,因B型主动脉夹层行血管腔内主动脉修复术(TEVAR)诱发的内脏动脉栓塞。在移植物缝线处上方的假腔(FL)盲袋内形成了巨大血栓。TEVAR扩张了真腔,FL血栓阻塞了起源于FL的内脏动脉。移植置换术后,FL袋内有血栓形成风险,且内脏动脉有血栓压迫风险。>