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原位继发龋病损发展的阈值间隙大小。

A threshold gap size for in situ secondary caries lesion development.

机构信息

Graduate Program in Dentistry, Federal University of Pelotas, Pelotas, RS, Brazil; Radboud University Medical Center, Department of Dentistry, Nijmegen, The Netherlands.

Radboud University Medical Center, Department of Dentistry, Nijmegen, The Netherlands.

出版信息

J Dent. 2019 Jan;80:36-40. doi: 10.1016/j.jdent.2018.10.014. Epub 2018 Nov 2.

Abstract

OBJECTIVES

This study investigated the influence of very small gaps in secondary caries (SC) development and additionally linked the threshold gap size with the caries activity level from volunteers.

METHODS

For 21 days, 15 volunteers wore a modified occlusal splint loaded with dentin-composite samples restored with different interfaces: bonded (B = samples restored with complete adhesive procedure), no-bonded (NB = restored with composite resin without adhesive procedure), and 30, 60 and 90 μm (no adhesive procedure and with intentional gap). The splint was dipped in a 20% sucrose solution (10 min) 8 x per day. Samples were imaged with transversal wavelength independent microradiography (T-WIM) and lesion depth and mineral loss were calculated. Average wall lesion depth from each volunteer was determined and according to the values the volunteers were grouped as high, mid and low caries activity levels.

RESULTS

No wall lesion formation was observed in B and NB groups. In general, intentional gaps led to SC lesion depth progression independent of caries activity level of volunteers. No substantial wall lesions were found for two volunteers. A trend for deeper lesion in larger gaps was observed for the high activity group.

CONCLUSION

Very small gaps around or wider than 30 μm develop SC independent of the caries activity level of the patient and SC wall lesion progression seemed to be related to individual factors even in this standardized in situ model.

SIGNIFICANCE

Independently of caries activity level of the patient, the threshold gap size for secondary caries wall lesion seems to be 30 μm at most.

摘要

目的

本研究调查了继发龋(SC)发展中非常小间隙的影响,并将阈值间隙大小与志愿者的龋病活动水平相关联。

方法

在 21 天内,15 名志愿者佩戴改良的咬合夹板,夹板上装载有不同界面的牙本质-复合树脂样本:粘结(B=用完整粘结程序修复的样本)、无粘结(NB=用无粘结程序的复合树脂修复的样本)和 30、60 和 90μm(无粘结程序和有意间隙)。夹板每天浸入 20%蔗糖溶液中(10 分钟)8 次。使用横向波长独立显微放射摄影术(T-WIM)对样本进行成像,并计算病变深度和矿物质损失。从每个志愿者确定平均壁病变深度,并根据这些值将志愿者分为高、中、低龋病活动水平。

结果

在 B 和 NB 组中未观察到壁病变形成。通常,有意间隙导致 SC 病变深度进展,而与志愿者的龋病活动水平无关。两名志愿者未发现实质性壁病变。对于高活性组,观察到较大间隙的病变深度更深的趋势。

结论

周围或大于 30μm 的非常小间隙会发展为 SC,而与患者的龋病活动水平无关,并且 SC 壁病变进展似乎与个体因素有关,即使在这种标准化的原位模型中也是如此。

意义

独立于患者的龋病活动水平,继发龋壁病变的阈值间隙大小最多似乎为 30μm。

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