Role of sendai virus fusion-glycoprotein in target cell susceptibility to cytotoxic T cells.

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超能文献

作者信息

Sugamura K, Shimizu K, Zarling D A, Bach F H

出版信息

Nature. 1977 Nov 17;270(5634):251-3. doi: 10.1038/270251a0.

DOI:10.1038/270251a0

PMID:304180

Abstract

摘要

相似文献

Role of sendai virus fusion-glycoprotein in target cell susceptibility to cytotoxic T cells.

Nature. 1977 Nov 17;270(5634):251-3. doi: 10.1038/270251a0.

Fusion of Sendai virus with the target cell membrane is required for T cell cytotoxicity.仙台病毒与靶细胞膜融合是T细胞细胞毒性所必需的。

Nature. 1978 Aug 17;274(5672):689-91. doi: 10.1038/274689a0.

Elicitation of anti-Sendai virus cytotoxic T lymphocytes by viral and H-2 antigens incorporated into the same lipid bilayer by membrane fusion and by reconstitution into liposomes.通过膜融合以及重组到脂质体中，将病毒和H-2抗原整合到同一脂质双层中，从而引发抗仙台病毒细胞毒性T淋巴细胞。

J Immunol. 1980 Feb;124(2):724-31.

Involvement of fusion activity of ultraviolet light-inactivated Sendai virus in formation of target antigens recognized by cytotoxic T cells.紫外线灭活仙台病毒的融合活性在细胞毒性T细胞识别的靶抗原形成中的作用。

J Exp Med. 1978 Jul 1;148(1):276-87. doi: 10.1084/jem.148.1.276.

Antigen-liposome modification of target cells as a method to alter their susceptibility to lysis by cytotoxic T lymphocytes.将抗原脂质体修饰靶细胞作为一种改变其对细胞毒性T淋巴细胞裂解敏感性的方法。

Proc Natl Acad Sci U S A. 1980 Oct;77(10):6105-8. doi: 10.1073/pnas.77.10.6105.

Elicitation of primary anti-Sendai virus cytotoxic T lymphocytes with purified viral glycoproteins.

Eur J Immunol. 1980 Dec;10(12):923-8. doi: 10.1002/eji.1830101207.

Stimulation of cytolytic T cells by isolated viral peptides and HN protein coupled to agarose beads.通过与琼脂糖珠偶联的分离病毒肽和HN蛋白刺激细胞溶解T细胞。

Nature. 1980 Jan 17;283(5744):308-11. doi: 10.1038/283308a0.

The induction of virus-specific cytotoxic T lymphocytes with solubilized viral and membrane proteins.用可溶性病毒蛋白和膜蛋白诱导病毒特异性细胞毒性T淋巴细胞。

J Exp Med. 1978 Dec 1;148(6):1620-7. doi: 10.1084/jem.148.6.1620.

Functional and structural considerations in the recognition of virus-infected cells by cytotoxic T lymphocytes.细胞毒性T淋巴细胞识别病毒感染细胞中的功能和结构考量

Contemp Top Mol Immunol. 1978;7:319-71. doi: 10.1007/978-1-4757-0779-3_10.

Recognition of viral glycoproteins by influenza A-specific cross-reactive cytolytic T lymphocytes.甲型流感特异性交叉反应性溶细胞性T淋巴细胞对病毒糖蛋白的识别

J Exp Med. 1980 Apr 1;151(4):945-58. doi: 10.1084/jem.151.4.945.

引用本文的文献

T-cell-mediated cytotoxic response to mumps virus in humans.人类对腮腺炎病毒的T细胞介导的细胞毒性反应。

Infect Immun. 1980 Oct;30(1):129-34. doi: 10.1128/iai.30.1.129-134.1980.

Cell-mediated cytotoxicity against targets bearing Sendai virus glycoproteins in the absence of viral infection.在无病毒感染情况下，针对携带仙台病毒糖蛋白的靶标的细胞介导细胞毒性。

J Virol. 1984 May;50(2):380-6. doi: 10.1128/JVI.50.2.380-386.1984.

Complement-dependent antiviral monospecific antibody-mediated lysis of murine cells coated with Sendai virus or its envelope component.补体依赖的抗病毒单特异性抗体介导的对涂有仙台病毒或其包膜成分的鼠细胞的裂解。

Infect Immun. 1980 Feb;27(2):355-63. doi: 10.1128/iai.27.2.355-363.1980.

Recognition of viral glycoproteins by influenza A-specific cross-reactive cytolytic T lymphocytes.甲型流感特异性交叉反应性溶细胞性T淋巴细胞对病毒糖蛋白的识别

J Exp Med. 1980 Apr 1;151(4):945-58. doi: 10.1084/jem.151.4.945.

Cytotoxic T-lymphocyte reactivity with individual Sendai virus glycoproteins.细胞毒性T淋巴细胞与仙台病毒各糖蛋白的反应性。

J Virol. 1985 Apr;54(1):53-7. doi: 10.1128/JVI.54.1.53-57.1985.

Cell-mediated immunity induced in mice after vaccination with a protease activation mutant, TR-2, of Sendai virus.用仙台病毒的蛋白酶激活突变体TR-2对小鼠进行疫苗接种后诱导的细胞介导免疫。

J Virol. 1988 Jul;62(7):2490-7. doi: 10.1128/JVI.62.7.2490-2497.1988.

Rotavirus-specific protein synthesis is not necessary for recognition of infected cells by virus-specific cytotoxic T lymphocytes.轮状病毒特异性蛋白合成对于病毒特异性细胞毒性T淋巴细胞识别受感染细胞并非必要。

J Virol. 1989 Aug;63(8):3279-83. doi: 10.1128/JVI.63.8.3279-3283.1989.

Difference in capacity of Sendai virus envelope components to induce cytotoxic T lymphocytes in primary and secondary immune responses.仙台病毒包膜成分在初次和二次免疫应答中诱导细胞毒性T淋巴细胞能力的差异。

Infect Immun. 1979 Dec;26(3):815-21. doi: 10.1128/iai.26.3.815-821.1979.

J Exp Med. 1978 Jul 1;148(1):276-87. doi: 10.1084/jem.148.1.276.

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