Nakashima Takashi, Takasugi Nobuhiro, Sahashi Yuki, Kubota Tomoki, Kawasaki Masanori
Department of Cardiology, Gifu University, Graduate School of Medicine, 1-1 Yanagido, Gifu 501-1194, Japan.
Department of Cardiology, Gifu University, Graduate School of Medicine, 1-1 Yanagido, Gifu 501-1194, Japan.
J Electrocardiol. 2019 Jan-Feb;52:63-65. doi: 10.1016/j.jelectrocard.2018.11.008. Epub 2018 Nov 6.
The role of left AV nodal (SVN) connections in the genesis of "left-variant" atypical atrioventricular nodal reentrant tachycardia (AVNRT) and those with multiple retrograde pathways remain unclear. We describe an unusual case of "left-variant" atypical AVNRT, where change in the retrograde earliest atrial activation site (REAAS) at the coronary sinus (CS) following radiofrequency catheter ablation (RFCA) was observed. Our observation suggests that the REAAS, that is, the left AVN connections, could participate in the formation of the reentrant circuit of "left-variant" atypical AVNRT. Furthermore, its atrial breakthroughs involved as a circuit of SVT could be (functionally) multiple.
左房室结（慢径）连接在“左侧变异型”非典型房室结折返性心动过速（AVNRT）及具有多条逆向传导路径的AVNRT发生机制中的作用仍不明确。我们描述了一例不寻常的“左侧变异型”非典型AVNRT病例，该病例在射频导管消融（RFCA）后，冠状静脉窦（CS）处的逆向最早心房激动部位（REAAS）发生了改变。我们的观察结果表明，REAAS，即左房室结连接，可能参与了“左侧变异型”非典型AVNRT折返环的形成。此外，其作为室上性心动过速环路的心房激动突破点可能（在功能上）是多个的。
