The Functional Heterogeneity of Resident Cardiac Macrophages in Myocardial Injury.
作者信息
Chen Bijun, Brickshawana Adipong, Frangogiannis Nikolaos G
机构信息
From the Department of Medicine (Cardiology), Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, NY.
出版信息
Circ Res. 2019 Jan 18;124(2):183-185. doi: 10.1161/CIRCRESAHA.118.314357.
Abstract
摘要
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本文引用的文献
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Tissue Resident CCR2- and CCR2+ Cardiac Macrophages Differentially Orchestrate Monocyte Recruitment and Fate Specification Following Myocardial Injury.组织驻留的 CCR2-和 CCR2+心脏巨噬细胞在心肌损伤后通过不同的方式募集单核细胞并决定其命运。
Circ Res. 2019 Jan 18;124(2):263-278. doi: 10.1161/CIRCRESAHA.118.314028.
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The human heart contains distinct macrophage subsets with divergent origins and functions.人类心脏中存在具有不同起源和功能的独特巨噬细胞亚群。
Nat Med. 2018 Aug;24(8):1234-1245. doi: 10.1038/s41591-018-0059-x. Epub 2018 Jun 11.
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Primitive Embryonic Macrophages are Required for Coronary Development and Maturation.冠状动脉发育和成熟需要原始胚胎巨噬细胞。
Circ Res. 2016 May 13;118(10):1498-511. doi: 10.1161/CIRCRESAHA.115.308270. Epub 2016 Mar 23.
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Revisiting Cardiac Cellular Composition.重新审视心脏细胞组成。
Circ Res. 2016 Feb 5;118(3):400-9. doi: 10.1161/CIRCRESAHA.115.307778. Epub 2015 Dec 3.
5
Distinct macrophage lineages contribute to disparate patterns of cardiac recovery and remodeling in the neonatal and adult heart.不同的巨噬细胞谱系对新生儿和成年心脏的心脏恢复和重塑的不同模式有贡献。
Proc Natl Acad Sci U S A. 2014 Nov 11;111(45):16029-34. doi: 10.1073/pnas.1406508111. Epub 2014 Oct 27.
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Differential contribution of monocytes to heart macrophages in steady-state and after myocardial infarction.在稳态和心肌梗死后，单核细胞对心脏巨噬细胞的贡献不同。
Circ Res. 2014 Jul 7;115(2):284-95. doi: 10.1161/CIRCRESAHA.115.303567. Epub 2014 May 1.
7
The inflammatory response in myocardial injury, repair, and remodelling.心肌损伤、修复及重塑中的炎症反应。
Nat Rev Cardiol. 2014 May;11(5):255-65. doi: 10.1038/nrcardio.2014.28. Epub 2014 Mar 25.
8
Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation.胚胎期和成年期来源的驻留心肌巨噬细胞在静息状态和炎症期间通过不同的机制维持。
Immunity. 2014 Jan 16;40(1):91-104. doi: 10.1016/j.immuni.2013.11.019.
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Myocardial ischemia/reperfusion injury is mediated by leukocytic toll-like receptor-2 and reduced by systemic administration of a novel anti-toll-like receptor-2 antibody.心肌缺血/再灌注损伤是由白细胞 Toll 样受体 2 介导的，而全身性给予新型抗 Toll 样受体 2 抗体可减轻损伤。
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Interleukin-1 receptor type I signaling critically regulates infarct healing and cardiac remodeling.I型白细胞介素-1受体信号传导对梗死愈合和心脏重塑起关键调节作用。
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