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Lineage choice decisions in B-cell development and leukemia.

作者信息

Raboso-Gallego Javier, Casado-García Ana, Rodríguez-Hernández Guillermo, Vicente-Dueñas Carolina, Sánchez-García Isidro

机构信息

Experimental Therapeutics and Translational Oncology Program, Instituto de Biología Molecular y Celular del Cáncer, CSIC-USAL, Campus M. de Unamuno s/n, Salamanca, Spain.

Institute of Biomedical Research of Salamanca (IBSAL), Salamanca, Spain.

出版信息

Stem Cell Investig. 2018 Dec 7;5:46. doi: 10.21037/sci.2018.12.01. eCollection 2018.

DOI:10.21037/sci.2018.12.01
PMID:30701181
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6327171/
Abstract
摘要

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本文引用的文献

1
T-cell leukemogenesis is an inappropriate lineage decision-making process: implications for precision oncology.
Mol Cell Oncol. 2018 Aug 7;5(4):e1497860. doi: 10.1080/23723556.2018.1497860. eCollection 2018.
2
The Making of Hematopoiesis: Developmental Ancestry and Environmental Nurture.造血的发生：发育起源与环境培育。
Int J Mol Sci. 2018 Jul 21;19(7):2122. doi: 10.3390/ijms19072122.
3
Lmo2 expression defines tumor cell identity during T-cell leukemogenesis.Lmo2 表达在 T 细胞白血病发生过程中定义肿瘤细胞特性。
EMBO J. 2018 Jul 13;37(14). doi: 10.15252/embj.201798783. Epub 2018 Jun 7.
4
Epigenetic Priming in Cancer Initiation.癌症起始中的表观遗传预激发
Trends Cancer. 2018 Jun;4(6):408-417. doi: 10.1016/j.trecan.2018.04.007.
5
The Making of Leukemia.白血病的形成。
Int J Mol Sci. 2018 May 17;19(5):1494. doi: 10.3390/ijms19051494.
6
Loss of Pax5 Exploits Sca1-BCR-ABL Susceptibility to Confer the Metabolic Shift Essential for pB-ALL.Pax5 缺失利用 Sca1-BCR-ABL 易感性导致代谢重编程，这对 pB-ALL 是必需的。
Cancer Res. 2018 May 15;78(10):2669-2679. doi: 10.1158/0008-5472.CAN-17-3262. Epub 2018 Feb 28.
7
Transcription factor Hoxb5 reprograms B cells into functional T lymphocytes.转录因子 Hoxb5 将 B 细胞重编程为功能性 T 淋巴细胞。
Nat Immunol. 2018 Mar;19(3):279-290. doi: 10.1038/s41590-018-0046-x. Epub 2018 Feb 12.
8
The genomic landscape of pediatric and young adult T-lineage acute lymphoblastic leukemia.儿童及青年T细胞系急性淋巴细胞白血病的基因组图谱
Nat Genet. 2017 Aug;49(8):1211-1218. doi: 10.1038/ng.3909. Epub 2017 Jul 3.
9
Infection Exposure Promotes Precursor B-cell Leukemia via Impaired H3K4 Demethylases.感染暴露通过损害 H3K4 去甲基化酶促进前 B 细胞白血病。
Cancer Res. 2017 Aug 15;77(16):4365-4377. doi: 10.1158/0008-5472.CAN-17-0701. Epub 2017 Jun 19.
10
Transcription factor EBF1 is essential for the maintenance of B cell identity and prevention of alternative fates in committed cells.转录因子 EBF1 对于维持 B 细胞特性和阻止定向细胞向其他命运转化是必不可少的。
Nat Immunol. 2013 Aug;14(8):867-75. doi: 10.1038/ni.2641. Epub 2013 Jun 30.