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Pertussis toxin-insensitive mechanism for carbachol-induced depolarization and positive inotropic effect in heart muscle.

作者信息

Pappano A J, Matsumoto K, Tajima T, Agnarsson U, Webb W

出版信息

Trends Pharmacol Sci. 1988 Feb;Suppl:35-9.

PMID:3074534
Abstract
摘要

相似文献

1
Pertussis toxin-insensitive mechanism for carbachol-induced depolarization and positive inotropic effect in heart muscle.
Trends Pharmacol Sci. 1988 Feb;Suppl:35-9.
2
A pharmacological investigation of the contribution of muscarinic receptor-linked potassium channels to the reversal by carbachol of positive inotropic responses of rabbit left atrium to cyclic AMP-generating agents.对毒蕈碱受体偶联钾通道在卡巴胆碱逆转兔左心房对环磷酸腺苷生成剂的正性肌力反应中所起作用的药理学研究。
J Pharmacol Exp Ther. 1993 Sep;266(3):1594-601.
3
Pertussis toxin-insensitive phosphoinositide hydrolysis, membrane depolarization, and positive inotropic effect of carbachol in chick atria.百日咳毒素不敏感的磷酸肌醇水解、膜去极化及卡巴胆碱对鸡心房的正性肌力作用
Circ Res. 1987 Sep;61(3):436-45. doi: 10.1161/01.res.61.3.436.
4
Positive vs. negative inotropic effects of carbachol in avian atrial muscle: role of Ni-like protein.
Circ Res. 1987 Oct;61(4 Pt 2):I105-11.
5
The action of pertussis toxin on heart adenylate cyclase and lipolysis in the parametrial adipose tissue of the rat.
Biomed Biochim Acta. 1987;46(8-9):S456-9.
6
Muscarinic agonist-induced positive inotropic response in chick atria.毒蕈碱激动剂诱导的鸡心房正性变力反应。
Life Sci. 1995;57(25):2365-76. doi: 10.1016/0024-3205(95)02231-7.
7
Carbachol depolarizes and accelerates pacemaker activity in the sinoatrial node of chicks treated with pertussis toxin.卡巴胆碱可使经百日咳毒素处理的雏鸡窦房结去极化并加速其起搏活动。
J Pharmacol Exp Ther. 1988 Oct;247(1):150-5.
8
Pretreatment with pertussis toxin blocks the protective effects of preconditioning: evidence for a G-protein mechanism.百日咳毒素预处理可阻断预处理的保护作用：G蛋白机制的证据。
J Mol Cell Cardiol. 1993 Mar;25(3):311-20. doi: 10.1006/jmcc.1993.1037.
9
Histamine H3-receptor signaling in the heart: possible involvement of Gi/Go proteins and N-type Ca++ channels.心脏中的组胺H3受体信号传导：Gi/Go蛋白和N型钙离子通道可能的参与情况。
J Pharmacol Exp Ther. 1994 Apr;269(1):221-9.
10
Pertussis toxin blocks adenosine A1 receptor mediated protection of the ischemic rat heart.百日咳毒素可阻断腺苷A1受体介导的对缺血大鼠心脏的保护作用。
J Mol Cell Cardiol. 1993 Jul;25(7):815-21. doi: 10.1006/jmcc.1993.1091.

引用本文的文献

1
Evidence for cocaine and methylecgonidine stimulation of M(2) muscarinic receptors in cultured human embryonic lung cells.可卡因和甲基爱康宁刺激培养的人胚肺细胞中M(2)毒蕈碱受体的证据。
Br J Pharmacol. 2001 Jan;132(2):451-60. doi: 10.1038/sj.bjp.0703819.
2
M1 muscarinic receptors increase calcium current and phosphoinositide turnover in guinea-pig ventricular cardiocytes.M1毒蕈碱受体可增加豚鼠心室心肌细胞的钙电流和磷酸肌醇代谢。
J Physiol. 1993 Nov;471:41-60. doi: 10.1113/jphysiol.1993.sp019890.
3
Agonists at mu-opioid, M2-muscarinic and GABAB-receptors increase the same potassium conductance in rat lateral parabrachial neurones.
μ-阿片受体、M2-毒蕈碱受体和GABAB受体的激动剂可增加大鼠臂旁外侧神经元的相同钾离子电导。
Br J Pharmacol. 1988 Nov;95(3):896-902. doi: 10.1111/j.1476-5381.1988.tb11719.x.
4
Pertussis toxin prevents adenosine receptor- and m-cholinoceptor-mediated sinus rate slowing and AV conduction block in the guinea-pig heart.百日咳毒素可防止豚鼠心脏中腺苷受体和毒蕈碱胆碱能受体介导的窦性心率减慢和房室传导阻滞。
Naunyn Schmiedebergs Arch Pharmacol. 1989 Jan-Feb;339(1-2):152-8. doi: 10.1007/BF00165137.
5
Sodium-dependent membrane current induced by carbachol in single guinea-pig ventricular myocytes.卡巴胆碱在单个豚鼠心室肌细胞中诱导的钠依赖性膜电流。
J Physiol. 1989 Aug;415:487-502. doi: 10.1113/jphysiol.1989.sp017733.
6
Phospholipase D in heart: basal activity and stimulation by phorbol esters and aluminum fluoride.
Naunyn Schmiedebergs Arch Pharmacol. 1992 Dec;346(6):607-13. doi: 10.1007/BF00168732.