Hwang T S, Park S H
Department of Pathology, College of Medicine, Seoul National University.
J Korean Med Sci. 1988 Mar;3(1):27-9. doi: 10.3346/jkms.1988.3.1.27.
We have reviewed 1066 thyroid lesions and compared the relative incidence of the so called 'palpation thyroiditis' between autoimmune thyroiditis and normal thyroid parenchyme surrounding the nodular thyroid lesion and also discussed the pathogenesis of palpation thyroiditis. The typical histopathologic features of 'palpation thyroiditis' were seen in 275 cases among 467 adenomatous goiters and in none of the autoimmune thyroiditis. We here in this paper suggest that the so called 'palpation thyroiditis' is not merely a secondary phenomenon to mechanical follicular damage by vigorous palpation, but this lesion more likely develops in conditions where certain types of physiologic alteration has occurred in follicular basement membrane, just like a pathogenesis of subacute granulomatous thyroiditis.
我们回顾了1066例甲状腺病变，比较了自身免疫性甲状腺炎与甲状腺结节性病变周围正常甲状腺实质中所谓“触诊性甲状腺炎”的相对发生率，并讨论了触诊性甲状腺炎的发病机制。在467例腺瘤性甲状腺肿中有275例出现了“触诊性甲状腺炎”的典型组织病理学特征，而自身免疫性甲状腺炎中无一例出现。我们在本文中提出，所谓的“触诊性甲状腺炎”并非仅仅是强力触诊导致机械性滤泡损伤的继发现象，而是这种病变更可能发生在滤泡基底膜出现某些生理性改变的情况下，就如同亚急性肉芽肿性甲状腺炎的发病机制一样。
