Ameghino Lucia, Rossi Malco, Merello Marcelo
Movement Disorders Section Neuroscience Department Raul Carrea Institute for Neurological Research (FLENI) Buenos Aires Argentina.
Argentine National Scientific and Technological Research Council (CONICET) Buenos Aires Argentina.
Mov Disord Clin Pract. 2016 Nov 25;3(6):626. doi: 10.1002/mdc3.12457. eCollection 2016 Nov-Dec.
Postural abnormalities in Parkinson's disease (PD) are considered the rule more than the exception and are disabling complications of the disease. These deformities include camptocormia, antecollis, Pisa syndrome, and scoliosis. Evidence to date suggests that postural deformities have a multifactorial pathophysiology, including muscular rigidity, axial dystonia, weakness due to myopathy, body scheme defects due to centrally impaired proprioception, and structural changes in the spine. Antecollis in parkinsonian disorders refers to a forward flexion of the head and neck. It is usually mild in severity and may be considered part of the stooped posture in patients with PD. Some authors that suggest the term antecollis should only be used in patients with at least a minimum of 45 degrees of thoracolumbar flexion. Neither camptocormia nor Pisa syndrome can be evaluated without taking into account the presence or absence of scoliosis. In this regard, the rotating component of the spine and its behavior in the supine position give important clues for a correct diagnosis. In some cases, X-rays in the standing and supine positions are necessary. The presence of marked camptocormia requires a minimum of flexion in the sagittal plane originating in the thoracolumbar spine greater than 45 degrees, with an almost complete resolution in the supine position. Pisa syndrome requires a minimum of 10 degrees of lateral flexion and is almost completely alleviated by passive mobilization or supine positioning. A certain degree of scoliosis is expected in most parkinsonian patients; therefore, both camptocormia and Pisa syndrome do not generally present as pure syndromes.
帕金森病(PD)中的姿势异常被认为是常见而非例外情况,并且是该疾病导致残疾的并发症。这些畸形包括脊柱前凸、颈前倾、比萨综合征和脊柱侧凸。迄今为止的证据表明,姿势畸形具有多因素病理生理学,包括肌肉僵硬、轴性肌张力障碍、肌病导致的无力、中枢性本体感觉受损引起的身体图式缺陷以及脊柱的结构变化。帕金森病性疾病中的颈前倾是指头颈部向前屈曲。其严重程度通常较轻,可能被视为帕金森病患者弯腰姿势的一部分。一些作者建议,颈前倾这个术语仅应用于胸腰椎屈曲至少达到45度的患者。在不考虑脊柱侧凸存在与否的情况下,无法评估脊柱前凸和比萨综合征。在这方面,脊柱的旋转成分及其在仰卧位的表现为正确诊断提供了重要线索。在某些情况下,站立位和仰卧位的X线检查是必要的。明显的脊柱前凸需要胸腰椎矢状面至少有大于45度的屈曲,且在仰卧位时几乎完全消失。比萨综合征需要至少10度的侧方屈曲,并且通过被动活动或仰卧位可几乎完全缓解。大多数帕金森病患者预计会有一定程度的脊柱侧凸;因此,脊柱前凸和比萨综合征通常不会以纯粹的综合征形式出现。