药物促进的B细胞耐受性诱导：环磷酰胺的一种依赖于非胸腺依赖性抗原耐受性的选择性活性。 - Suppr | 超能文献

Drug-promoted B cell tolerance induction: a selective activity of cyclophosphamide dependent on the tolerogenicity of thymus-independent antigens.

作者信息

Howard J G, Hale C

出版信息

Eur J Immunol. 1978 Jul;8(7):492-6. doi: 10.1002/eji.1830080708.

DOI:10.1002/eji.1830080708

Abstract

摘要

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Drug-promoted B cell tolerance induction: a selective activity of cyclophosphamide dependent on the tolerogenicity of thymus-independent antigens.药物促进的B细胞耐受性诱导：环磷酰胺的一种依赖于非胸腺依赖性抗原耐受性的选择性活性。

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The nature of drug-induced B cell tolerance.药物诱导的B细胞耐受性的本质。

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Patterns of isotype expression by B cell clones responding to thymus-dependent and thymus-independent antigens in vitro.体外对胸腺依赖性和胸腺非依赖性抗原作出反应的B细胞克隆的同种型表达模式。

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Induction and persistence of B-cell tolerance to the thymus-dependent component of the alpha(1 leads to 6) glucosyl determinant of dextran. Recovery induced by treatment with dextranase in vivo.对葡聚糖α(1→6)葡萄糖基决定簇的胸腺依赖性成分的B细胞耐受性的诱导与维持。体内用葡聚糖酶治疗诱导的恢复。

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Immunological tolerance affects only a subpopulation of the antigen-specific B lymphocytes: evidence against clonal deletion as the mechanism of tolerance induction.免疫耐受仅影响抗原特异性B淋巴细胞的一个亚群：反对克隆清除作为耐受诱导机制的证据。

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Clin Exp Immunol. 1981 Apr;44(1):74-81.