Diacono J, Dietrich J, Lajoix H
C R Acad Sci III. 1986;302(14):533-6.
In the rat atrium, the contraction rebound occurring after ACh treatment could not be related to an increase in action potential duration and disappeared in the presence of atropine or caffeine. Contractures elicited by caffeine (10 and 20 mM) were inhibited by ACh. This inhibition was suppressed by atropine. The hypothesis that ACh could increase the sarcoplasmic reticulum Ca diastolic reuptake is discussed.
在大鼠心房中，乙酰胆碱（ACh）处理后出现的收缩反弹与动作电位持续时间的增加无关，且在存在阿托品或咖啡因的情况下消失。咖啡因（10和20 mM）引发的挛缩被ACh抑制。这种抑制作用被阿托品抑制。文中讨论了ACh可能增加肌浆网钙舒张期再摄取的假说。
